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The Journal of Experimental Medicine|July 16, 2017
PD-L1 up-regulation restrains Th17 cell differentiation in STAT3 loss- and STAT1 gain-of-function patientsYuan Zhang, Chi A Ma, Monica G Lawrence, et al.Pediatric Allergy and Immunology : Official Publication of the European Society of Pediatric Allergy and Immunology|October 24, 2020
Novel PGM3 compound heterozygous variants with IgE-related dermatitis, lymphopenia, without syndromic featuresAna García-García, Monserrat Buendia Arellano, Àngela Deyà-Martínez, et al.The Journal of Allergy and Clinical Immunology|July 28, 2020
Heritable risk for severe anaphylaxis associated with increased α-tryptase-encoding germline copy number at TPSAB1Jonathan J Lyons, Jack Chovanec, Michael P O'Connell, et al.Blood|April 18, 2024
Standardized indolent systemic mastocytosis evaluations across a health care system: implications for screening accuracyJeremy C McMurray, Curtis S Pacheco, Brandon J Schornack, et al.The Journal of Clinical Investigation|September 20, 2016
FOXP3+ Tregs require WASP to restrain Th2-mediated food allergyWillem S Lexmond, Jeremy A Goettel, Jonathan J Lyons, et al.Journal of Human Immunity|January 29, 2026
Extended clinical phenotypes and long-term outcomes of phosphoglucomutase-3 deficiencyChen Wang, Meera Patel, Amanda Urban, et al.Genetics in Medicine : Official Journal of the American College of Medical Genetics|July 9, 2016
Prospective phenotyping of NGLY1-CDDG, the first congenital disorder of deglycosylationChristina Lam, Carlos Ferreira, Donna Krasnewich, et al.Allergy|March 13, 2024
High burden of clonal mast cell disorders and hereditary α-tryptasemia in patients who need Hymenoptera venom immunotherapyPeter Korošec, Gunter J Sturm, Jonathan J Lyons, et al.The Journal of Allergy and Clinical Immunology|June 8, 2024
Mast cell activation syndrome: Current understanding and research needsMariana Castells, Matthew P Giannetti, Matthew J Hamilton, et al.The Journal of Allergy and Clinical Immunology|March 5, 2014
Autosomal recessive phosphoglucomutase 3 (PGM3) mutations link glycosylation defects to atopy, immune deficiency, autoimmunity, and neurocognitive impairmentYu Zhang, Xiaomin Yu, Mie Ichikawa, et al.Pageof 9