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Journal of Cellular Physiology|March 5, 2008
Alpha-1 adrenergic receptor transactivates signal transducer and activator of transcription-3 (Stat3) through activation of Src and epidermal growth factor receptor (EGFR) in hepatocytesChang Han, William C Bowen, George K Michalopoulos, et al.
Molecular Cancer Therapeutics|November 6, 2009
Omega-3 polyunsaturated fatty acids inhibit hepatocellular carcinoma cell growth through blocking beta-catenin and cyclooxygenase-2Kyu Lim, Chang Han, Yifan Dai, et al.
World Journal of Hepatology|April 8, 2015
MiR-122 in hepatitis B virus and hepatitis C virus dual infectionKyoungsub Song, Chang Han, Srikanta Dash, et al.
The American Journal of Pathology|June 10, 2014
miR-185 inhibits hepatocellular carcinoma growth by targeting the DNMT1/PTEN/Akt pathwayXimena V Qadir, Chang Han, Dongdong Lu, et al.
Cancer Research|February 19, 2015
Omega-3 Polyunsaturated Fatty Acids Upregulate 15-PGDH Expression in Cholangiocarcinoma Cells by Inhibiting miR-26a/b ExpressionLu Yao, Chang Han, Kyoungsub Song, et al.
The American Journal of Pathology|March 22, 2015
Deletion of Mir155 prevents Fas-induced liver injury through up-regulation of Mcl-1Weina Chen, Chang Han, Jinqiang Zhang, et al.
Plos One|July 22, 2015
miR-150 Deficiency Protects against FAS-Induced Acute Liver Injury in Mice through Regulation of AKTWeina Chen, Chang Han, Jinqiang Zhang, et al.
American Journal of Physiology. Gastrointestinal and Liver Physiology|March 19, 2002
85-kDa cPLA(2) plays a critical role in PPAR-mediated gene transcription in human hepatoma cellsChang Han, A Jake Demetris, George Michalopoulos, et al.
Hepatology (Baltimore, Md.)|July 9, 2009
Cyclooxygenase-2 prevents fas-induced liver injury through up-regulation of epidermal growth factor receptorGuiying Li, Chang Han, Lihong Xu, et al.
Hepatology (Baltimore, Md.)|August 27, 2003
Cyclooxygenase-2 promotes hepatocellular carcinoma cell growth through Akt activation: evidence for Akt inhibition in celecoxib-induced apoptosisJing Leng, Chang Han, A Jake Demetris, et al.
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