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Nature Genetics|June 1, 2005
Gains of glycosylation comprise an unexpectedly large group of pathogenic mutationsGuillaume Vogt, Ariane Chapgier, Kun Yang, et al.Npj Aging|August 24, 2023
Severe COVID-19 patients exhibit elevated levels of autoantibodies targeting cardiolipin and platelet glycoprotein with age: a systems biology approachDennyson Leandro M Fonseca, Igor Salerno Filgueiras, Alexandre H C Marques, et al.Blood|February 4, 2012
B cell-intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in miceMike Recher, Siobhan O Burns, Miguel A de la Fuente, et al.Nature Communications|December 8, 2018
GPCR-specific autoantibody signatures are associated with physiological and pathological immune homeostasisOtavio Cabral-Marques, Alexandre Marques, Lasse Melvær Giil, et al.Journal of Medical Virology|February 1, 2023
Cross-sectional analysis reveals autoantibody signatures associated with COVID-19 severityGabriela C Baiocchi, Aristo Vojdani, Avi Z Rosenberg, et al.The Journal of Allergy and Clinical Immunology|March 9, 2018
CD40 ligand deficiency causes functional defects of peripheral neutrophils that are improved by exogenous IFN-γOtavio Cabral-Marques, Tabata Takahashi França, Ashraf Al-Sbiei, et al.JCI Insight|June 7, 2019
Safety and early efficacy outcomes for lentiviral fibroblast gene therapy in recessive dystrophic epidermolysis bullosaSu M Lwin, Farhatullah Syed, Wei-Li Di, et al.The Journal of Experimental Medicine|June 26, 2019
Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responsesSarah Spencer, Sevgi Köstel Bal, William Egner, et al.Nature Communications|March 10, 2022
Autoantibodies targeting GPCRs and RAS-related molecules associate with COVID-19 severityOtavio Cabral-Marques, Gilad Halpert, Lena F Schimke, et al.The Journal of Allergy and Clinical Immunology|June 12, 2017
Hematopoietic stem cell transplantation in patients with gain-of-function signal transducer and activator of transcription 1 mutationsJennifer W Leiding, Satoshi Okada, David Hagin, et al.Pageof 52