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Hans Hombauer

Showing results (11-20 of 19) with videos related to

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Molecular Cell|July 2, 2014
PCNA and Msh2-Msh6 activate an Mlh1-Pms1 endonuclease pathway required for Exo1-independent mismatch repairEva M Goellner, Catherine E Smith, Christopher S Campbell, et al.
Nature Communications|November 3, 2020
Extensive 5'-surveillance guards against non-canonical NAD-caps of nuclear mRNAs in yeastYaqing Zhang, David Kuster, Tobias Schmidt, et al.
Plos Genetics|November 9, 2013
Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an exonuclease 1-independent mismatch repair pathwayCatherine E Smith, Marc L Mendillo, Nikki Bowen, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 19, 2017
Alterations in cellular metabolism triggered by <i>URA7</i> or <i>GLN3</i> inactivation cause imbalanced dNTP pools and increased mutagenesisTobias T Schmidt, Gloria Reyes, Kerstin Gries, et al.
Nucleic Acids Research|October 25, 2019
Inactivation of folylpolyglutamate synthetase Met7 results in genome instability driven by an increased dUTP/dTTP ratioTobias T Schmidt, Sushma Sharma, Gloria X Reyes, et al.
Current Biology : CB|January 8, 2021
Ligation of newly replicated DNA controls the timing of DNA mismatch repairGloria X Reyes, Anna Kolodziejczak, Lovely Jael Paul Solomon Devakumar, et al.
Nucleic Acids Research|November 22, 2018
A genetic screen pinpoints ribonucleotide reductase residues that sustain dNTP homeostasis and specifies a highly mutagenic type of dNTP imbalanceTobias T Schmidt, Sushma Sharma, Gloria X Reyes, et al.
Current Biology : CB|December 5, 2003
Identification of a subunit of a novel Kleisin-beta/SMC complex as a potential substrate of protein phosphatase 2AFoong May Yeong, Hans Hombauer, Kerstin S Wendt, et al.
Cell|July 20, 2010
SRD5A3 is required for converting polyprenol to dolichol and is mutated in a congenital glycosylation disorderVincent Cantagrel, Dirk J Lefeber, Bobby G Ng, et al.
Pageof 2

Showing results (11-20 of 19) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 19 results.
Molecular Cell|July 2, 2014
PCNA and Msh2-Msh6 activate an Mlh1-Pms1 endonuclease pathway required for Exo1-independent mismatch repairEva M Goellner, Catherine E Smith, Christopher S Campbell, et al.
Nature Communications|November 3, 2020
Extensive 5'-surveillance guards against non-canonical NAD-caps of nuclear mRNAs in yeastYaqing Zhang, David Kuster, Tobias Schmidt, et al.
Plos Genetics|November 9, 2013
Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an exonuclease 1-independent mismatch repair pathwayCatherine E Smith, Marc L Mendillo, Nikki Bowen, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 19, 2017
Alterations in cellular metabolism triggered by <i>URA7</i> or <i>GLN3</i> inactivation cause imbalanced dNTP pools and increased mutagenesisTobias T Schmidt, Gloria Reyes, Kerstin Gries, et al.
Nucleic Acids Research|October 25, 2019
Inactivation of folylpolyglutamate synthetase Met7 results in genome instability driven by an increased dUTP/dTTP ratioTobias T Schmidt, Sushma Sharma, Gloria X Reyes, et al.
Current Biology : CB|January 8, 2021
Ligation of newly replicated DNA controls the timing of DNA mismatch repairGloria X Reyes, Anna Kolodziejczak, Lovely Jael Paul Solomon Devakumar, et al.
Nucleic Acids Research|November 22, 2018
A genetic screen pinpoints ribonucleotide reductase residues that sustain dNTP homeostasis and specifies a highly mutagenic type of dNTP imbalanceTobias T Schmidt, Sushma Sharma, Gloria X Reyes, et al.
Current Biology : CB|December 5, 2003
Identification of a subunit of a novel Kleisin-beta/SMC complex as a potential substrate of protein phosphatase 2AFoong May Yeong, Hans Hombauer, Kerstin S Wendt, et al.
Cell|July 20, 2010
SRD5A3 is required for converting polyprenol to dolichol and is mutated in a congenital glycosylation disorderVincent Cantagrel, Dirk J Lefeber, Bobby G Ng, et al.
Pageof 2