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Hans Lassmann

Showing results (311-320 of 373) with videos related to

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EMBO Reports|June 16, 2007
Mutation in the Scyl1 gene encoding amino-terminal kinase-like protein causes a recessive form of spinocerebellar neurodegenerationWolfgang M Schmidt, Cornelia Kraus, Harald Höger, et al.
Journal of Neuroinflammation|October 27, 2017
Human antibodies against the myelin oligodendrocyte glycoprotein can cause complement-dependent demyelinationPatrick Peschl, Kathrin Schanda, Bleranda Zeka, et al.
Nature Communications|November 3, 2018
Acute microglia ablation induces neurodegeneration in the somatosensory systemStephen J Rubino, Lior Mayo, Isabella Wimmer, et al.
Acta Neuropathologica Communications|December 26, 2013
T cell-activation in neuromyelitis optica lesions plays a role in their formationMaria Pohl, Naoto Kawakami, Maja Kitic, et al.
Journal of Neuropathology and Experimental Neurology|May 16, 2017
Oxidative Injury and Iron Redistribution Are Pathological Hallmarks of Marmoset Experimental Autoimmune EncephalomyelitisJordon Dunham, Jan Bauer, Graham R Campbell, et al.
Frontiers in Immunology|December 17, 2020
Mannan-MOG35-55 Reverses Experimental Autoimmune Encephalomyelitis, Inducing a Peripheral Type 2 Myeloid Response, Reducing CNS Inflammation, and Preserving Axons in Spinal Cord LesionsAnastasia Dagkonaki, Maria Avloniti, Maria Evangelidou, et al.
Brain : a Journal of Neurology|June 4, 2018
Impaired plasticity of macrophages in X-linked adrenoleukodystrophyIsabelle Weinhofer, Bettina Zierfuss, Simon Hametner, et al.
Acta Neuropathologica|November 5, 2015
Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNSBleranda Zeka, Maria Hastermann, Sonja Hochmeister, et al.
Development (Cambridge, England)|January 23, 2014
A single allele of Hdac2 but not Hdac1 is sufficient for normal mouse brain development in the absence of its paralogAstrid Hagelkruys, Sabine Lagger, Julia Krahmer, et al.
Annals of Neurology|June 27, 2009
Antibodies produced by clonally expanded plasma cells in multiple sclerosis cerebrospinal fluidGregory P Owens, Jeffrey L Bennett, Hans Lassmann, et al.
Pageof 38

Showing results (311-320 of 373) with videos related to

Sort By:
Pageof 38
EMBO Reports|June 16, 2007
Mutation in the Scyl1 gene encoding amino-terminal kinase-like protein causes a recessive form of spinocerebellar neurodegenerationWolfgang M Schmidt, Cornelia Kraus, Harald Höger, et al.
Journal of Neuroinflammation|October 27, 2017
Human antibodies against the myelin oligodendrocyte glycoprotein can cause complement-dependent demyelinationPatrick Peschl, Kathrin Schanda, Bleranda Zeka, et al.
Nature Communications|November 3, 2018
Acute microglia ablation induces neurodegeneration in the somatosensory systemStephen J Rubino, Lior Mayo, Isabella Wimmer, et al.
Acta Neuropathologica Communications|December 26, 2013
T cell-activation in neuromyelitis optica lesions plays a role in their formationMaria Pohl, Naoto Kawakami, Maja Kitic, et al.
Journal of Neuropathology and Experimental Neurology|May 16, 2017
Oxidative Injury and Iron Redistribution Are Pathological Hallmarks of Marmoset Experimental Autoimmune EncephalomyelitisJordon Dunham, Jan Bauer, Graham R Campbell, et al.
Frontiers in Immunology|December 17, 2020
Mannan-MOG35-55 Reverses Experimental Autoimmune Encephalomyelitis, Inducing a Peripheral Type 2 Myeloid Response, Reducing CNS Inflammation, and Preserving Axons in Spinal Cord LesionsAnastasia Dagkonaki, Maria Avloniti, Maria Evangelidou, et al.
Brain : a Journal of Neurology|June 4, 2018
Impaired plasticity of macrophages in X-linked adrenoleukodystrophyIsabelle Weinhofer, Bettina Zierfuss, Simon Hametner, et al.
Acta Neuropathologica|November 5, 2015
Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNSBleranda Zeka, Maria Hastermann, Sonja Hochmeister, et al.
Development (Cambridge, England)|January 23, 2014
A single allele of Hdac2 but not Hdac1 is sufficient for normal mouse brain development in the absence of its paralogAstrid Hagelkruys, Sabine Lagger, Julia Krahmer, et al.
Annals of Neurology|June 27, 2009
Antibodies produced by clonally expanded plasma cells in multiple sclerosis cerebrospinal fluidGregory P Owens, Jeffrey L Bennett, Hans Lassmann, et al.
Pageof 38