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Nature Communications|July 17, 2019
Patient-specific cancer genes contribute to recurrently perturbed pathways and establish therapeutic vulnerabilities in esophageal adenocarcinomaThanos P Mourikis, Lorena Benedetti, Elizabeth Foxall, et al.Nature Communications|November 29, 2022
Genetically personalised organ-specific metabolic models in health and diseaseCarles Foguet, Yu Xu, Scott C Ritchie, et al.Molecular Cancer|January 5, 2013
Analysis of colorectal cancers in British Bangladeshi identifies early onset, frequent mucinous histotype and a high prevalence of RBFOX1 deletionNeel Sengupta, Christopher Yau, Anuratha Sakthianandeswaren, et al.Frontiers in Oncology|July 21, 2022
Patient Derived Organoids Confirm That PI3K/AKT Signalling Is an Escape Pathway for Radioresistance and a Target for Therapy in Rectal CancerKasun Wanigasooriya, Joao D Barros-Silva, Louise Tee, et al.Statistics in Medicine|July 15, 2025
Incorporation of Patient and Public Involvement in Statistical Methodology Research: Summary of Workshop ProceedingsAiden Smith, Hannah Worboys, Samina Begum, et al.Nature Communications|July 13, 2026
Transcriptomic analysis of tissue-resident memory T cells of the fallopian tube reveals a precursor immune surveillance network for ovarian cancer preventionLuyao Wang, Breeshey Roskams-Hieter, Nosheen Hussain, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|January 15, 2021
The Oxford Classic Links Epithelial-to-Mesenchymal Transition to Immunosuppression in Poor Prognosis Ovarian CancersZhiyuan Hu, Paula Cunnea, Zhe Zhong, et al.BMJ Open|October 30, 2021
Protocol for the development of a core outcome set for studies of pregnant women with pre-existing multimorbiditySiang Ing Lee, Kelly-Ann Eastwood, Ngawai Moss, et al.British Journal of Cancer|March 30, 2021
Promises and challenges of adoptive T-cell therapies for solid tumoursMatteo Morotti, Ashwag Albukhari, Abdulkhaliq Alsaadi, et al.The American Journal of Gastroenterology|September 18, 2013
Survival in stage II/III colorectal cancer is independently predicted by chromosomal and microsatellite instability, but not by specific driver mutationsDmitri Mouradov, Enric Domingo, Peter Gibbs, et al.Pageof 10