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The Journal of Allergy and Clinical Immunology|July 8, 2008
Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced T(H)17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndromeEllen D Renner, Stacey Rylaarsdam, Stephanie Anover-Sombke, et al.American Journal of Human Genetics|October 22, 2013
Germline mutations in NFKB2 implicate the noncanonical NF-κB pathway in the pathogenesis of common variable immunodeficiencyKarin Chen, Emily M Coonrod, Attila Kumánovics, et al.The Journal of Allergy and Clinical Immunology|September 7, 2010
Diagnostic approach to the hyper-IgE syndromes: immunologic and clinical key findings to differentiate hyper-IgE syndromes from atopic dermatitisLena F Schimke, Julie Sawalle-Belohradsky, Joachim Roesler, et al.Blood Cells, Molecules & Diseases|August 24, 2010
Hematologically important mutations: X-linked chronic granulomatous disease (third update)Dirk Roos, Douglas B Kuhns, Anne Maddalena, et al.Cancer Immunology, Immunotherapy : CII|December 27, 2011
Exploiting antitumor immunity to overcome relapse and improve remission durationLei L Chen, Xinjian Chen, Haesun Choi, et al.The Journal of Allergy and Clinical Immunology. in Practice|March 17, 2019
Outcomes and Treatment Strategies for Autoimmunity and Hyperinflammation in Patients with RAG DeficiencyJocelyn R Farmer, Zsofia Foldvari, Boglarka Ujhazi, et al.Pageof 11