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Human Molecular Genetics|November 9, 2013
New Lmna knock-in mice provide a molecular mechanism for the 'segmental aging' in Hutchinson-Gilford progeria syndromeHea-Jin Jung, Yiping Tu, Shao H Yang, et al.
Gut Microbes|April 25, 2024
Intestinal carbapenem-resistant <i>Klebsiella pneumoniae</i> undergoes complex transcriptional reprogramming following immune activationClement David, Aleksander Czauderna, Liqing Cheng, et al.
Molecular and Cellular Biology|October 15, 2014
An absence of nuclear lamins in keratinocytes leads to ichthyosis, defective epidermal barrier function, and intrusion of nuclear membranes and endoplasmic reticulum into the nuclear chromatinHea-Jin Jung, Angelica Tatar, Yiping Tu, et al.
Molecular Biology of the Cell|March 28, 2014
Reciprocal knock-in mice to investigate the functional redundancy of lamin B1 and lamin B2John M Lee, Yiping Tu, Angelica Tatar, et al.
The Journal of Clinical Investigation|March 22, 2016
Modulation of LMNA splicing as a strategy to treat prelamin A diseasesJohn M Lee, Chika Nobumori, Yiping Tu, et al.
Scientific Reports|December 3, 2016
Multiparameter mechanical and morphometric screening of cellsMahdokht Masaeli, Dewal Gupta, Sean O'Byrne, et al.
Molecular Biology of the Cell|October 7, 2011
Deficiencies in lamin B1 and lamin B2 cause neurodevelopmental defects and distinct nuclear shape abnormalities in neuronsCatherine Coffinier, Hea-Jin Jung, Chika Nobumori, et al.
Human Molecular Genetics|February 6, 2015
Mice that express farnesylated versions of prelamin A in neurons develop achalasiaShao H Yang, Shiri Procaccia, Hea-Jin Jung, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 8, 2013
Farnesylation of lamin B1 is important for retention of nuclear chromatin during neuronal migrationHea-Jin Jung, Chika Nobumori, Chris N Goulbourne, et al.
Annals of Neurology|May 1, 2016
PYCR2 Mutations cause a lethal syndrome of microcephaly and failure to thriveMaha S Zaki, Gifty Bhat, Tipu Sultan, et al.
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