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Catheterization and Cardiovascular Interventions : Official Journal of the Society for Cardiac Angiography & Interventions|June 2, 2025
Long-Term Follow-Up After Direct-Flow Transcatheter Aortic Valve Implantation: A Single Center ExperienceDan M Prunea, Raffael Geissler, Alexandru Achim, et al.
Cardiovascular Research|July 17, 2024
Beneficial normalization of cardiac repolarization by carnitine in transgenic short QT syndrome type 1 rabbit modelsIlona Bodi, Lea Mettke, Konstantin Michaelides, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|October 4, 2021
Cardiomyocyte-specific miR-100 overexpression preserves heart function under pressure overload in mice and diminishes fatty acid uptake as well as ROS production by direct suppression of Nox4 and CD36Christian Smolka, Delia Schlösser, Christoph Koentges, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|October 3, 2022
Functional remodelling of perinuclear mitochondria alters nucleoplasmic Ca2+ signalling in heart failureJulia Voglhuber, Michael Holzer, Snježana Radulović, et al.
Plos One|January 24, 2013
Mechanistic target of rapamycin (Mtor) is essential for murine embryonic heart development and growthYi Zhu, Karla M P Pires, Kevin J Whitehead, et al.
Europace : European Pacing, Arrhythmias, and Cardiac Electrophysiology : Journal of the Working Groups on Cardiac Pacing, Arrhythmias, and Cardiac Cellular Electrophysiology of the European Society of Cardiology|April 3, 2019
Postpartum hormones oxytocin and prolactin cause pro-arrhythmic prolongation of cardiac repolarization in long QT syndrome type 2Ilona Bodi, Jonathan Sorge, Alessandro Castiglione, et al.
Basic Research in Cardiology|May 13, 2015
SIRT3 deficiency impairs mitochondrial and contractile function in the heartChristoph Koentges, Katharina Pfeil, Tilman Schnick, et al.
Journal of Molecular and Cellular Cardiology|June 16, 2019
Impaired SIRT3 activity mediates cardiac dysfunction in endotoxemia by calpain-dependent disruption of ATP synthesisChristoph Koentges, María C Cimolai, Katharina Pfeil, et al.
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