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Journal of Cell Communication and Signaling|December 2, 2017
Tumor cell-intrinsic phenotypic plasticity facilitates adaptive cellular reprogramming driving acquired drug resistanceHeinz Hammerlindl, Helmut SchaiderClinical Epigenetics|March 10, 2019
Emerging roles of H3K9me3, SETDB1 and SETDB2 in therapy-induced cellular reprogrammingJoachim Torrano, Abdullah Al Emran, Heinz Hammerlindl, et al.Theranostics|June 3, 2020
Epigenetics and metabolism at the crossroads of stress-induced plasticity, stemness and therapeutic resistance in cancerDinoop Ravindran Menon, Heinz Hammerlindl, Joachim Torrano, et al.Science Advances|June 12, 2026
ResMap: A community resource for systematic mapping of therapy-persistent residual cancer cell dependencies across contextsXiaoxiao Sun, Savitha Gayathri, Karl Kumbier, et al.Nature Communications|March 19, 2025
A phenopushing platform to identify compounds that alleviate acute hypoxic stress by fast-tracking cellular adaptationLi Li, Heinz Hammerlindl, Susan Q Shen, et al.Nature Biotechnology|July 11, 2025
Transitive prediction of small-molecule function through alignment of high-content screening resourcesFeng Bao, Li Li, Heinz Hammerlindl, et al.Cancer Medicine|February 23, 2019
Magnolol induces cell death through PI3K/Akt-mediated epigenetic modifications boosting treatment of BRAF- and NRAS-mutant melanomaAbdullah Al Emran, Brinda Reddy Chinna Chowdary, Farzana Ahmed, et al.Epigenomics|May 10, 2019
Commonly integrated epigenetic modifications of differentially expressed genes lead to adaptive resistance in cancerAbdullah Al Emran, Diego M Marzese, Dinoop R Menon, et al.Cancer Research|January 24, 2016
Notch4 Signaling Induces a Mesenchymal-Epithelial-like Transition in Melanoma Cells to Suppress Malignant BehaviorsEhsan Bonyadi Rad, Heinz Hammerlindl, Christian Wels, et al.Science Advances|January 28, 2022
Modulating environmental signals to reveal mechanisms and vulnerabilities of cancer persistersXiaoxiao Sun, Jake M Bieber, Heinz Hammerlindl, et al.Pageof 2