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Helena M Viola

Showing results (1-10 of 43) with videos related to

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Journal of Molecular and Cellular Cardiology|August 7, 2010
Qo site of mitochondrial complex III is the source of increased superoxide after transient exposure to hydrogen peroxideHelena M Viola, Livia C Hool
Current Drug Targets|February 5, 2011
Targeting calcium and the mitochondria in prevention of pathology in the heartHelena M Viola, Livia C Hool
Heart, Lung & Circulation|March 25, 2014
How does calcium regulate mitochondrial energetics in the heart? - new insightsHelena M Viola, Livia C Hool
Archives of Biochemistry and Biophysics|March 20, 2019
Impaired calcium handling and mitochondrial metabolic dysfunction as early markers of hypertrophic cardiomyopathyHelena M Viola, Livia C Hool
Elife|July 11, 2017
Auto-regulation in the powerhouseHelena M Viola, Livia C Hool
Clinical and Experimental Pharmacology & Physiology|August 13, 2009
Cross-talk between L-type Ca2+ channels and mitochondriaHelena M Viola, Livia C Hool
Clinical and Experimental Pharmacology & Physiology|April 5, 2013
Role of the cytoskeleton in communication between L-type Ca(2+) channels and mitochondriaHelena M Viola, Livia C Hool
Circulation Research|March 10, 2007
Transient exposure to hydrogen peroxide causes an increase in mitochondria-derived superoxide as a result of sustained alteration in L-type Ca2+ channel function in the absence of apoptosis in ventricular myocytesHelena M Viola, Peter G Arthur, Livia C Hool
Journal of Molecular and Cellular Cardiology|January 27, 2009
Evidence for regulation of mitochondrial function by the L-type Ca2+ channel in ventricular myocytesHelena M Viola, Peter G Arthur, Livia C Hool
Genes|March 25, 2017
Dystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel TherapiesVictoria P A Johnstone, Helena M Viola, Livia C Hool
Pageof 5

Showing results (1-10 of 43) with videos related to

Sort By:
Pageof 5
Journal of Molecular and Cellular Cardiology|August 7, 2010
Qo site of mitochondrial complex III is the source of increased superoxide after transient exposure to hydrogen peroxideHelena M Viola, Livia C Hool
Current Drug Targets|February 5, 2011
Targeting calcium and the mitochondria in prevention of pathology in the heartHelena M Viola, Livia C Hool
Heart, Lung & Circulation|March 25, 2014
How does calcium regulate mitochondrial energetics in the heart? - new insightsHelena M Viola, Livia C Hool
Archives of Biochemistry and Biophysics|March 20, 2019
Impaired calcium handling and mitochondrial metabolic dysfunction as early markers of hypertrophic cardiomyopathyHelena M Viola, Livia C Hool
Elife|July 11, 2017
Auto-regulation in the powerhouseHelena M Viola, Livia C Hool
Clinical and Experimental Pharmacology & Physiology|August 13, 2009
Cross-talk between L-type Ca2+ channels and mitochondriaHelena M Viola, Livia C Hool
Clinical and Experimental Pharmacology & Physiology|April 5, 2013
Role of the cytoskeleton in communication between L-type Ca(2+) channels and mitochondriaHelena M Viola, Livia C Hool
Circulation Research|March 10, 2007
Transient exposure to hydrogen peroxide causes an increase in mitochondria-derived superoxide as a result of sustained alteration in L-type Ca2+ channel function in the absence of apoptosis in ventricular myocytesHelena M Viola, Peter G Arthur, Livia C Hool
Journal of Molecular and Cellular Cardiology|January 27, 2009
Evidence for regulation of mitochondrial function by the L-type Ca2+ channel in ventricular myocytesHelena M Viola, Peter G Arthur, Livia C Hool
Genes|March 25, 2017
Dystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel TherapiesVictoria P A Johnstone, Helena M Viola, Livia C Hool
Pageof 5