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Nature Communications|March 21, 2024
Structure of an open KATP channel reveals tandem PIP2 binding sites mediating the Kir6.2 and SUR1 regulatory interfaceCamden M Driggers, Yi-Ying Kuo, Phillip Zhu, et al.
Biorxiv : the Preprint Server for Biology|August 14, 2023
Structure of an open K ATP channel reveals tandem PIP 2 binding sites mediating the Kir6.2 and SUR1 regulatory interfaceCamden M Driggers, Yi-Ying Kuo, Phillip Zhu, et al.
Pediatric Diabetes|March 13, 2019
Functional characterization of activating mutations in the sulfonylurea receptor 1 (ABCC8) causing neonatal diabetes mellitus in Asian Indian childrenKandasamy Balamurugan, Babu Kavitha, Zhongying Yang, et al.
Channels (Austin, Tex.)|March 15, 2024
Dynamic duo: Kir6 and SUR in KATP channel structure and functionBruce L Patton, Phillip Zhu, Assmaa ElSheikh, et al.
The Journal of Biological Chemistry|August 4, 2017
NMDA receptors mediate leptin signaling and regulate potassium channel trafficking in pancreatic β-cellsYi Wu, Dale A Fortin, Veronica A Cochrane, et al.
The Journal of Biological Chemistry|December 25, 2009
Characterization and functional restoration of a potassium channel Kir6.2 pore mutation identified in congenital hyperinsulinismJeremy D Bushman, Joel W Gay, Paul Tewson, et al.
The Journal of General Physiology|February 16, 2011
N-terminal transmembrane domain of SUR1 controls gating of Kir6.2 by modulating channel sensitivity to PIP2Emily B Pratt, Paul Tewson, Cathrin E Bruederle, et al.
The Journal of Biological Chemistry|February 28, 2002
Identification of a familial hyperinsulinism-causing mutation in the sulfonylurea receptor 1 that prevents normal trafficking and function of KATP channelsGrit Taschenberger, Adam Mougey, Shu Shen, et al.
The Journal of Biological Chemistry|February 1, 2015
Structurally distinct ligands rescue biogenesis defects of the KATP channel complex via a converging mechanismPrasanna K Devaraneni, Gregory M Martin, Erik M Olson, et al.
The Journal of Biological Chemistry|January 20, 2009
Sulfonylurea receptor 1 mutations that cause opposite insulin secretion defects with chemical chaperone exposureEmily B Pratt, Fei-Fei Yan, Joel W Gay, et al.
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