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Molecular Endocrinology (Baltimore, Md.)|December 13, 2005
Repulsive separation of the cytoplasmic ends of transmembrane helices 3 and 6 is linked to receptor activation in a novel thyrotropin receptor mutant (M626I)Usanee Ringkananont, Joost Van Durme, Lucia Montanelli, et al.
Immunology Letters|September 1, 2017
The effect of CT26 tumor-derived TGF-β on the balance of tumor growth and immunityStephanie Y Owyang, Min Zhang, Grace A Walkup, et al.
The Journal of Clinical Endocrinology and Metabolism|February 26, 2009
Loss-of-function mutations in the thyrotropin receptor gene as a major determinant of hyperthyrotropinemia in a consanguineous communityYardena Tenenbaum-Rakover, Helmut Grasberger, Sunee Mamanasiri, et al.
Human Genetics|September 29, 2005
Identification of a locus for nongoitrous congenital hypothyroidism on chromosome 15q25.3-26.1Helmut Grasberger, Martine Vaxillaire, Silvana Pannain, et al.
The Journal of Biological Chemistry|January 16, 2009
Activation of dual oxidases Duox1 and Duox2: differential regulation mediated by camp-dependent protein kinase and protein kinase C-dependent phosphorylationSabrina Rigutto, Candice Hoste, Helmut Grasberger, et al.
Helicobacter|October 7, 2020
Dendritic cell-derived TGF-β mediates the induction of mucosal regulatory T-cell response to Helicobacter infection essential for maintenance of immune tolerance in miceStephanie Y Owyang, Min Zhang, Mohamad El-Zaatari, et al.
Redox Biology|December 9, 2016
Deficiency in Duox2 activity alleviates ileitis in GPx1- and GPx2-knockout mice without affecting apoptosis incidence in the crypt epitheliumFong-Fong Chu, R Steven Esworthy, James H Doroshow, et al.
The Journal of Clinical Endocrinology and Metabolism|November 29, 2007
Biallelic inactivation of the dual oxidase maturation factor 2 (DUOXA2) gene as a novel cause of congenital hypothyroidismIlaria Zamproni, Helmut Grasberger, Francesca Cortinovis, et al.
The Journal of Clinical Endocrinology and Metabolism|February 9, 2006
A novel thyroid hormone receptor-beta mutation that fails to bind nuclear receptor corepressor in a patient as an apparent cause of severe, predominantly pituitary resistance to thyroid hormoneSharon Y Wu, Ronald N Cohen, Enver Simsek, et al.
Scientific Reports|July 7, 2018
Increased risk for inflammatory bowel disease in congenital hypothyroidism supports the existence of a shared susceptibility factorHelmut Grasberger, Mohamed Noureldin, Timothy D Kao, et al.
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