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The Journal of Heart and Lung Transplantation : the Official Publication of the International Society for Heart Transplantation|April 6, 2004
The structural examination of myocardial samples from patients with end-stage heart failure supported by ventricular assist devices using electron microscopy and amino acid analysis reveals low degree of reverse remodelingHendrik Milting, Monika Jacob, Astrid Kassner, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|November 7, 2007
Up-regulation of nestin in the infarcted myocardium potentially indicates differentiation of resident cardiac stem cells into various lineages including cardiomyocytesSergiu Scobioala, Rainer Klocke, Michael Kuhlmann, et al.
Cellular and Molecular Life Sciences : CMLS|November 14, 2025
The ARVC-5-associated protein TMEM43 controls mitochondrial energy metabolism by stabilising ER-mitochondrial contact sitesKai Jürgens, Lena Menzel, Nora Klinke, et al.
Journal of Cardiovascular Development and Disease|July 25, 2022
Pathogenic Variants in Cardiomyopathy Disorder Genes Underlie Pediatric Myocarditis-Further Impact of Heterozygous Immune Disorder Gene Variants?Franziska Seidel, Kai Thorsten Laser, Karin Klingel, et al.
Basic Research in Cardiology|September 1, 2009
K201 improves aspects of the contractile performance of human failing myocardium via reduction in Ca2+ leak from the sarcoplasmic reticulumKarl Toischer, Stephan E Lehnart, Gero Tenderich, et al.
The Journal of Heart and Lung Transplantation : the Official Publication of the International Society for Heart Transplantation|October 20, 2009
Ventricular unloading is associated with increased 20s proteasome protein expression in the myocardiumJeremias Wohlschlaeger, Stephan Urs Sixt, Tatjana Stoeppler, et al.
Human Mutation|May 12, 2017
The novel αB-crystallin (CRYAB) mutation p.D109G causes restrictive cardiomyopathyAndreas Brodehl, Anna Gaertner-Rommel, Bärbel Klauke, et al.
Basic Research in Cardiology|November 11, 2011
G-CSF therapy reduces myocardial repolarization reserve in the presence of increased arteriogenesis, angiogenesis and connexin 43 expression in an experimental model of pacing-induced heart failurePeter Milberg, Rainer Klocke, Gerrit Frommeyer, et al.
Circulation|August 17, 2005
Increased cardiac expression of tissue inhibitor of metalloproteinase-1 and tissue inhibitor of metalloproteinase-2 is related to cardiac fibrosis and dysfunction in the chronic pressure-overloaded human heartStephane Heymans, Blanche Schroen, Pieter Vermeersch, et al.
The Journal of Heart and Lung Transplantation : the Official Publication of the International Society for Heart Transplantation|April 1, 2008
Decreased myocardial chromogranin a expression and colocalization with brain natriuretic peptide during reverse cardiac remodeling after ventricular unloadingJeremias Wohlschlaeger, Moritz von Winterfeld, Hendrik Milting, et al.
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