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Cancer Letters|June 24, 2022
Mechanisms used by cancer cells to tolerate drug-induced replication stressHendrika A Segeren, Bart WestendorpSTAR Protocols|August 17, 2021
Collection of cells for single-cell RNA sequencing using high-resolution fluorescence microscopyHendrika A Segeren, Kiki C Andree, Lisa Oomens, et al.Cell|February 1, 2025
Long-term imaging of individual ribosomes reveals ribosome cooperativity in mRNA translationMaximilian F Madern, Sora Yang, Olivier Witteveen, et al.Oncogene|April 8, 2022
Oncogenic RAS sensitizes cells to drug-induced replication stress via transcriptional silencing of P53Hendrika A Segeren, Elsbeth A van Liere, Frank M Riemers, et al.Molecular Oncology|February 26, 2025
Stochastic variation in the FOXM1 transcription program mediates replication stress toleranceHendrika A Segeren, Kathryn A Wierenga, Frank M Riemers, et al.The EMBO Journal|September 3, 2019
Cyclin F-dependent degradation of E2F7 is critical for DNA repair and G2-phase progressionRuixue Yuan, Qingwu Liu, Hendrika A Segeren, et al.Cell Reports|December 2, 2020
Excessive E2F Transcription in Single Cancer Cells Precludes Transient Cell-Cycle Exit after DNA DamageHendrika A Segeren, Lotte M van Rijnberk, Eva Moreno, et al.EMBO Reports|February 17, 2016
Feedback regulation between atypical E2Fs and APC/CCdh1 coordinates cell cycle progressionMichiel Boekhout, Ruixue Yuan, Annelotte P Wondergem, et al.Cancer Research|March 5, 2022
Genome-Protective Topoisomerase 2a-Dependent G2 Arrest Requires p53 in hTERT-Positive Cancer CellsNicola Lockwood, Silvia Martini, Ainara Lopez-Pardo, et al.Pageof 1