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Bioorganic Chemistry|September 11, 2020
Inhibition of zika virus infection by fused tricyclic derivatives of 1,2,4,5-tetrahydroimidazo[1,5-a]quinolin-3(3aH)-oneBin Xu, Emily M Lee, Angelica Medina, et al.
Plos Pathogens|April 13, 2012
Productive hepatitis C virus infection of stem cell-derived hepatocytes reveals a critical transition to viral permissiveness during differentiationXianfang Wu, Jason M Robotham, Emily Lee, et al.
Analytical Chemistry|January 27, 2010
Simultaneous reduction and digestion of proteins with disulfide bonds for hydrogen/deuterium exchange monitored by mass spectrometryHui-Min Zhang, Shaun M McLoughlin, Stephen D Frausto, et al.
The Journal of Biological Chemistry|April 22, 2009
The isomerase active site of cyclophilin A is critical for hepatitis C virus replicationUdayan Chatterji, Michael Bobardt, Suganya Selvarajah, et al.
Antimicrobial Agents and Chemotherapy|May 20, 2009
Cyclosporine inhibits flavivirus replication through blocking the interaction between host cyclophilins and viral NS5 proteinMin Qing, Feng Yang, Bo Zhang, et al.
Journal of Virology|March 10, 2025
A single mutation in the PrM gene of Zika virus determines AXL dependency for infection of human neural cellsRenu Khasa, Sarah C Ogden, Yuqing Wang, et al.
PNAS Nexus|May 19, 2025
Interferon-dependent R-loop induction by Zika virus contributes to growth attenuationYijing Zhao, Anna D Metzler, Yangping Li, et al.
Journal of Virology|May 16, 2014
Cell death-inducing DFFA-like effector b is required for hepatitis C virus entry into hepatocytesXianfang Wu, Emily M Lee, Christy Hammack, et al.
Nucleic Acids Research|September 5, 2009
Short-hairpin RNAs delivered by lentiviral vector transduction trigger RIG-I-mediated IFN activationRachael Kenworthy, Diana Lambert, Feng Yang, et al.
Hepatology (Baltimore, Md.)|June 3, 2009
Mutations in the hepatitis C virus polymerase that increase RNA binding can confer resistance to cyclosporine AZhe Liu, John M Robida, Sreedhar Chinnaswamy, et al.
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