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Methods in Molecular Medicine|July 13, 2005
Gene silencing using adenoviral RNAi vector in vascular smooth muscle cells and cardiomyocytesHideko Kasahara, Hiroki AokiCardiovascular Research|September 15, 2004
Biochemical analyses of eight NKX2.5 homeodomain missense mutations causing atrioventricular block and cardiac anomaliesHideko Kasahara, D Woodrow BensonDevelopmental Dynamics : an Official Publication of the American Association of Anatomists|January 5, 2020
Defective coronary vessel organization and reduction of VEGF-A in mouse embryonic hearts with gestational mild hypoxiaLawrence X Cai, Fariz F Alkassis, Hideko KasaharaJBMR Plus|December 22, 2023
The Association between Sleep and Bone Mineral Density: Cross-Sectional Study Using Health Check-up Data in a Local Hospital in JapanReiko Yamaura, Hideko Kasahara, Satoshi Iimuro, et al.Acta Crystallographica. Section F, Structural Biology and Crystallization Communications|November 11, 2008
Crystallization and preliminary X-ray analysis of the NKX2.5 homeodomain in complex with DNACaroli Genis, Peyton Scone, Hideko Kasahara, et al.Molecular and Cellular Biology|December 4, 2003
Myocardin expression is regulated by Nkx2.5, and its function is required for cardiomyogenesisTomomi Ueyama, Hideko Kasahara, Takahiro Ishiwata, et al.The Journal of Biological Chemistry|May 20, 2003
Csm, a cardiac-specific isoform of the RNA helicase Mov10l1, is regulated by Nkx2.5 in embryonic heartTomomi Ueyama, Hideko Kasahara, Takahiro Ishiwata, et al.Journal of Cardiovascular Electrophysiology|July 26, 2002
Developmentally modulated cardiac conduction failure in transgenic mice with fetal or postnatal overexpression of DNA nonbinding mutant Nkx2.5Hiroko Wakimoto, Hideko Kasahara, Colin T Maguire, et al.Vascular Health and Risk Management|May 15, 2026
Longitudinal Associations Between Metabolic Risk Burden and the Cardio-Ankle Vascular Index (CAVI) in Japan Using Health-Check CohortYuzo Horibuchi, Larbi Aluariachy, Reiko Yamaura, et al.Molecular Biology of the Cell|August 20, 2003
A novel variant of cardiac myosin-binding protein-C that is unable to assemble into sarcomeres is expressed in the aged mouse atriumNaruki Sato, Tsutomu Kawakami, Ayako Nakayama, et al.Pageof 5