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Plos Genetics|December 21, 2020
CFAP53 regulates mammalian cilia-type motility patterns through differential localization and recruitment of axonemal dynein componentsTakahiro Ide, Wang Kyaw Twan, Hao Lu, et al.
Science Advances|August 4, 2020
Role of Ca2+ transients at the node of the mouse embryo in breaking of left-right symmetryKatsutoshi Mizuno, Kei Shiozawa, Takanobu A Katoh, et al.
Science (New York, N.Y.)|September 18, 2012
Cilia at the node of mouse embryos sense fluid flow for left-right determination via Pkd2Satoko Yoshiba, Hidetaka Shiratori, Ivana Y Kuo, et al.
Plos One|October 16, 2015
Single-Cell Expression Profiling Reveals a Dynamic State of Cardiac Precursor Cells in the Early Mouse EmbryoIoannis Kokkinopoulos, Hidekazu Ishida, Rie Saba, et al.
Human Molecular Genetics|January 15, 2010
Dysregulation of the PDGFRA gene causes inflow tract anomalies including TAPVR: integrating evidence from human genetics and model organismsSteven B Bleyl, Yukio Saijoh, Noortje A M Bax, et al.
Developmental Cell|March 16, 2017
A Wnt5 Activity Asymmetry and Intercellular Signaling via PCP Proteins Polarize Node Cells for Left-Right Symmetry BreakingKatsura Minegishi, Masakazu Hashimoto, Rieko Ajima, et al.
Neuron|April 25, 2020
Loss of PYCR2 Causes Neurodegeneration by Increasing Cerebral Glycine Levels via SHMT2Nathalie Escande-Beillard, Abigail Loh, Sahar N Saleem, et al.
American Journal of Human Genetics|August 4, 2016
TTC25 Deficiency Results in Defects of the Outer Dynein Arm Docking Machinery and Primary Ciliary Dyskinesia with Left-Right Body Asymmetry RandomizationJulia Wallmeier, Hidetaka Shiratori, Gerard W Dougherty, et al.
Cell|March 14, 2007
An Nkx2-5/Bmp2/Smad1 negative feedback loop controls heart progenitor specification and proliferationOwen W J Prall, Mary K Menon, Mark J Solloway, et al.
Nature Communications|November 3, 2020
CFAP45 deficiency causes situs abnormalities and asthenospermia by disrupting an axonemal adenine nucleotide homeostasis moduleGerard W Dougherty, Katsutoshi Mizuno, Tabea Nöthe-Menchen, et al.
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