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Hideyuki Yamada

Showing results (61-70 of 101) with videos related to

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Cancer Medicine|May 10, 2023
Less necessity of adjuvant S-1 treatment in non-monarchE-eligible patientsMuhan Yu, Mamoru Takada, Hideyuki Yamada, et al.
Biochemical Pharmacology|May 14, 2018
The aryl hydrocarbon receptor is indispensable for dioxin-induced defects in sexually-dimorphic behaviors due to the reduction in fetal steroidogenesis of the pituitary-gonadal axis in ratsYukiko Hattori, Tomoki Takeda, Arisa Nakamura, et al.
Archives of Biochemistry and Biophysics|June 8, 2002
Activation of microsomal epoxide hydrolase by interaction with cytochromes P450: kinetic analysis of the association and substrate-specific activation of epoxide hydrolase functionKen-ichiro Taura Ki, Hideyuki Yamada, Eri Naito, et al.
The Journal of Toxicological Sciences|April 7, 2011
2,3,7,8-tetrachlorodibenzo-p-dioxin potentially attenuates the gene expression of pituitary gonadotropin β-subunits in a fetal age-specific fashion: a comparative study using cultured pituitariesTomoki Takeda, Midori Yamamoto, Masaru Himeno, et al.
Life Sciences|February 13, 2007
Suppression of fetal testicular cytochrome P450 17 by maternal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin: a mechanism involving an initial effect on gonadotropin synthesis in the pituitaryJunko Taketoh, Junpei Mutoh, Tomoki Takeda, et al.
Drug Metabolism and Disposition: the Biological Fate of Chemicals|July 10, 2014
A useful model capable of predicting the clearance of cytochrome 3A4 (CYP3A4) substrates in humans: validity of CYP3A4 transgenic mice lacking their own Cyp3a enzymesTetsuya Mitsui, Takayuki Nemoto, Taiji Miyake, et al.
Molecular Pharmacology|October 18, 2013
Maternal exposure to dioxin imprints sexual immaturity of the pups through fixing the status of the reduced expression of hypothalamic gonadotropin-releasing hormoneTomoki Takeda, Misaki Fujii, Yukiko Hattori, et al.
The Journal of Toxicological Sciences|January 11, 2017
α-Lipoic acid potentially targets AMP-activated protein kinase and energy production in the fetal brain to ameliorate dioxin-produced attenuation in fetal steroidogenesisTomoki Takeda, Yuki Matsuo, Kyoko Nishida, et al.
The Journal of Toxicological Sciences|March 20, 2015
Dehydroepiandrosterone sulfate and cytochrome P450 inducers alleviate fatty liver in male rats fed an orotic acid-supplemented dietKenji Takeuchi, Hirohiko Goto, Yuko Ito, et al.
The Journal of Toxicological Sciences|November 13, 2015
Effect of in utero exposure to endocrine disruptors on fetal steroidogenesis governed by the pituitary-gonad axis: a study in rats using different ways of administrationYudai Kariyazono, Junki Taura, Yukiko Hattori, et al.
Pageof 11

Showing results (61-70 of 101) with videos related to

Sort By:
Pageof 11
Cancer Medicine|May 10, 2023
Less necessity of adjuvant S-1 treatment in non-monarchE-eligible patientsMuhan Yu, Mamoru Takada, Hideyuki Yamada, et al.
Biochemical Pharmacology|May 14, 2018
The aryl hydrocarbon receptor is indispensable for dioxin-induced defects in sexually-dimorphic behaviors due to the reduction in fetal steroidogenesis of the pituitary-gonadal axis in ratsYukiko Hattori, Tomoki Takeda, Arisa Nakamura, et al.
Archives of Biochemistry and Biophysics|June 8, 2002
Activation of microsomal epoxide hydrolase by interaction with cytochromes P450: kinetic analysis of the association and substrate-specific activation of epoxide hydrolase functionKen-ichiro Taura Ki, Hideyuki Yamada, Eri Naito, et al.
The Journal of Toxicological Sciences|April 7, 2011
2,3,7,8-tetrachlorodibenzo-p-dioxin potentially attenuates the gene expression of pituitary gonadotropin β-subunits in a fetal age-specific fashion: a comparative study using cultured pituitariesTomoki Takeda, Midori Yamamoto, Masaru Himeno, et al.
Life Sciences|February 13, 2007
Suppression of fetal testicular cytochrome P450 17 by maternal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin: a mechanism involving an initial effect on gonadotropin synthesis in the pituitaryJunko Taketoh, Junpei Mutoh, Tomoki Takeda, et al.
Drug Metabolism and Disposition: the Biological Fate of Chemicals|July 10, 2014
A useful model capable of predicting the clearance of cytochrome 3A4 (CYP3A4) substrates in humans: validity of CYP3A4 transgenic mice lacking their own Cyp3a enzymesTetsuya Mitsui, Takayuki Nemoto, Taiji Miyake, et al.
Molecular Pharmacology|October 18, 2013
Maternal exposure to dioxin imprints sexual immaturity of the pups through fixing the status of the reduced expression of hypothalamic gonadotropin-releasing hormoneTomoki Takeda, Misaki Fujii, Yukiko Hattori, et al.
The Journal of Toxicological Sciences|January 11, 2017
α-Lipoic acid potentially targets AMP-activated protein kinase and energy production in the fetal brain to ameliorate dioxin-produced attenuation in fetal steroidogenesisTomoki Takeda, Yuki Matsuo, Kyoko Nishida, et al.
The Journal of Toxicological Sciences|March 20, 2015
Dehydroepiandrosterone sulfate and cytochrome P450 inducers alleviate fatty liver in male rats fed an orotic acid-supplemented dietKenji Takeuchi, Hirohiko Goto, Yuko Ito, et al.
The Journal of Toxicological Sciences|November 13, 2015
Effect of in utero exposure to endocrine disruptors on fetal steroidogenesis governed by the pituitary-gonad axis: a study in rats using different ways of administrationYudai Kariyazono, Junki Taura, Yukiko Hattori, et al.
Pageof 11