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Hiroyasu Nakano

Showing results (71-80 of 128) with videos related to

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Journal of General Internal Medicine|May 6, 2024
Anti-TIF1-γ Antibody-Positive Dermatomyositis Leading to Identification of Diffuse Large B-Cell LymphomaTakuya Otsuki, Kosuke Ishizuka, Hiromitsu Eto, et al.
Nihon Shokakibyo Gakkai Zasshi = the Japanese Journal of Gastro-Enterology|June 7, 2016
A case of Ménétrier's disease seemingly caused by hilar cholangiocarcinomaNozomi Sato, Kazunari Nakahara, Ryo Morita, et al.
Plos Biology|May 10, 2019
Regulation of membrane phospholipid asymmetry by Notch-mediated flippase expression controls the number of intraepithelial TCRαβ+CD8αα+ T cellsChieko Ishifune, Shin-Ichi Tsukumo, Yoichi Maekawa, et al.
FEBS Letters|December 25, 2007
Rac, PAK and p38 regulate cell contact-dependent nuclear translocation of myocardin-related transcription factorAttila Sebe, András Masszi, Matthew Zulys, et al.
Journal of Neurochemistry|February 19, 2013
Tumor necrosis factor receptor-associated factor 5 is an essential mediator of ischemic brain infarctionLang Wang, Yanyun Lu, Hongjing Guan, et al.
Mucosal Immunology|August 23, 2022
IκBζ controls IL-17-triggered gene expression program in intestinal epithelial cells that restricts colonization of SFB and prevents Th17-associated pathologiesSoh Yamazaki, Naohiro Inohara, Masaki Ohmuraya, et al.
American Journal of Physiology. Cell Physiology|October 12, 2012
Hyperosmotic stress regulates the distribution and stability of myocardin-related transcription factor, a key modulator of the cytoskeletonDonald L Ly, Faiza Waheed, Monika Lodyga, et al.
The Journal of Biological Chemistry|August 19, 2005
Tumor necrosis factor alpha (TNFalpha) induces the unfolded protein response (UPR) in a reactive oxygen species (ROS)-dependent fashion, and the UPR counteracts ROS accumulation by TNFalphaXin Xue, Jiang-Hu Piao, Akihito Nakajima, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 3, 2007
TRAF-1, -2, -3, -5, and -6 are induced in atherosclerotic plaques and differentially mediate proinflammatory functions of CD40L in endothelial cellsAndreas Zirlik, Udo Bavendiek, Peter Libby, et al.
The EMBO Journal|July 26, 2003
NF-kappaB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell deathSachiko Sakon, Xin Xue, Mutsuhiro Takekawa, et al.
Pageof 13

Showing results (71-80 of 128) with videos related to

Sort By:
Pageof 13
Journal of General Internal Medicine|May 6, 2024
Anti-TIF1-γ Antibody-Positive Dermatomyositis Leading to Identification of Diffuse Large B-Cell LymphomaTakuya Otsuki, Kosuke Ishizuka, Hiromitsu Eto, et al.
Nihon Shokakibyo Gakkai Zasshi = the Japanese Journal of Gastro-Enterology|June 7, 2016
A case of Ménétrier's disease seemingly caused by hilar cholangiocarcinomaNozomi Sato, Kazunari Nakahara, Ryo Morita, et al.
Plos Biology|May 10, 2019
Regulation of membrane phospholipid asymmetry by Notch-mediated flippase expression controls the number of intraepithelial TCRαβ+CD8αα+ T cellsChieko Ishifune, Shin-Ichi Tsukumo, Yoichi Maekawa, et al.
FEBS Letters|December 25, 2007
Rac, PAK and p38 regulate cell contact-dependent nuclear translocation of myocardin-related transcription factorAttila Sebe, András Masszi, Matthew Zulys, et al.
Journal of Neurochemistry|February 19, 2013
Tumor necrosis factor receptor-associated factor 5 is an essential mediator of ischemic brain infarctionLang Wang, Yanyun Lu, Hongjing Guan, et al.
Mucosal Immunology|August 23, 2022
IκBζ controls IL-17-triggered gene expression program in intestinal epithelial cells that restricts colonization of SFB and prevents Th17-associated pathologiesSoh Yamazaki, Naohiro Inohara, Masaki Ohmuraya, et al.
American Journal of Physiology. Cell Physiology|October 12, 2012
Hyperosmotic stress regulates the distribution and stability of myocardin-related transcription factor, a key modulator of the cytoskeletonDonald L Ly, Faiza Waheed, Monika Lodyga, et al.
The Journal of Biological Chemistry|August 19, 2005
Tumor necrosis factor alpha (TNFalpha) induces the unfolded protein response (UPR) in a reactive oxygen species (ROS)-dependent fashion, and the UPR counteracts ROS accumulation by TNFalphaXin Xue, Jiang-Hu Piao, Akihito Nakajima, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 3, 2007
TRAF-1, -2, -3, -5, and -6 are induced in atherosclerotic plaques and differentially mediate proinflammatory functions of CD40L in endothelial cellsAndreas Zirlik, Udo Bavendiek, Peter Libby, et al.
The EMBO Journal|July 26, 2003
NF-kappaB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell deathSachiko Sakon, Xin Xue, Mutsuhiro Takekawa, et al.
Pageof 13