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Biological Trace Element Research|April 21, 2015
A Potential New Mechanism of Arsenic Carcinogenesis: Depletion of Stem-Loop Binding Protein and Increase in Polyadenylated Canonical Histone H3.1 mRNAJason Brocato, Danqi Chen, Jianli Liu, et al.Biochemistry|April 3, 2009
Heterochromatinization as a potential mechanism of nickel-induced carcinogenesisThomas P Ellen, Thomas Kluz, Mark E Harder, et al.Heliyon|September 1, 2022
Loss of MEG3 and upregulation of miR-145 play an important role in the invasion and migration of Cr(VI)-transformed cellsZhuo Zhang, Jingxia Li, Bo Yan, et al.Environmental Health Perspectives|November 14, 2002
Enhanced overexpression of an HIF-1/hypoxia-related protein in cancer cellsHakan Cangul, Konstantin Salnikow, Herman Yee, et al.Molecular Medicine Reports|July 18, 2017
Protective effects of marrubiin improve endometriosis through suppression of the expression of RANTESXiao-Hong SunIscience|September 13, 2020
Polyadenylation of Histone H3.1 mRNA Promotes Cell Transformation by Displacing H3.3 from Gene Regulatory ElementsDanqi Chen, Qiao Yi Chen, Zhenjia Wang, et al.Cancer Research|July 4, 2003
The involvement of hypoxia-inducible transcription factor-1-dependent pathway in nickel carcinogenesisKonstantin Salnikow, Todd Davidson, Qunwei Zhang, et al.Journal of Cellular Physiology|September 17, 2008
Soluble and insoluble nickel compounds exert a differential inhibitory effect on cell growth through IKKalpha-dependent cyclin D1 down-regulationWeiming Ouyang, Dongyun Zhang, Jingxia Li, et al.Molecular Carcinogenesis|May 2, 2006
Soluble nickel inhibits HIF-prolyl-hydroxylases creating persistent hypoxic signaling in A549 cellsTodd L Davidson, Haobin Chen, Dominic M Di Toro, et al.Dalton Transactions (Cambridge, England : 2003)|January 13, 2010
Nickel binding to histone H4Maria Antonietta Zoroddu, Massimiliano Peana, Serenella Medici, et al.Pageof 282