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Biochemical and Biophysical Research Communications|May 24, 2015
Pannexin 1 deficiency can induce hearing lossHong-Bo Zhao, Yan Zhu, Chun Liang, et al.Biochemical and Biophysical Research Communications|April 16, 2014
Deafness induced by Connexin 26 (GJB2) deficiency is not determined by endocochlear potential (EP) reduction but is associated with cochlear developmental disordersJin Chen, Jing Chen, Yan Zhu, et al.International Journal of Molecular Sciences|May 2, 2018
Knockout of Pannexin-1 Induces Hearing LossJin Chen, Chun Liang, Liang Zong, et al.Scientific Reports|August 25, 2017
Corrigendum: Pannexin1 channels dominate ATP release in the cochlea ensuring endocochlear potential and auditory receptor potential generation and hearingJin Chen, Yan Zhu, Chun Liang, et al.Scientific Reports|June 3, 2015
Pannexin1 channels dominate ATP release in the cochlea ensuring endocochlear potential and auditory receptor potential generation and hearingJin Chen, Yan Zhu, Chun Liang, et al.Nature Communications|May 9, 2013
Active cochlear amplification is dependent on supporting cell gap junctionsYan Zhu, Chun Liang, Jin Chen, et al.Neurobiology of Disease|August 22, 2017
A deafness mechanism of digenic Cx26 (GJB2) and Cx30 (GJB6) mutations: Reduction of endocochlear potential by impairment of heterogeneous gap junctional function in the cochlear lateral wallLing Mei, Jin Chen, Liang Zong, et al.Journal of Neurophysiology|December 15, 2021
Efferent neurons control hearing sensitivity and protect hearing from noise through the regulation of gap junctions between cochlear supporting cellsHong-Bo Zhao, Li-Man Liu, Ning Yu, et al.Biochemical and Biophysical Research Communications|May 30, 2017
Progressive age-dependence and frequency difference in the effect of gap junctions on active cochlear amplification and hearingLiang Zong, Jin Chen, Yan Zhu, et al.Biorxiv : the Preprint Server for Biology|July 3, 2026
Upregulation of ATP-purinergic P2x2 receptors in the cochlea over-amplifies hearing sensitivity leading to hyperacusis and attenuation by antagonistsTian-Ying Zhai, Chun Liang, Jin Chen, et al.Pageof 831