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The European Journal of Neuroscience|May 5, 2005
Connexin26 is responsible for anionic molecule permeability in the cochlea for intercellular signalling and metabolic communicationsHong-Bo ZhaoFrontiers in Molecular Neuroscience|June 13, 2017
Hypothesis of K+-Recycling Defect Is Not a Primary Deafness Mechanism for Cx26 (GJB2) DeficiencyHong-Bo ZhaoBMC Cell Biology|May 28, 2016
Expression and function of pannexins in the inner ear and hearingHong-Bo ZhaoThe Journal of Comparative Neurology|September 26, 2006
Distinct and gradient distributions of connexin26 and connexin30 in the cochlear sensory epithelium of guinea pigsHong-Bo Zhao, Ning YuPurinergic Signalling|September 1, 2010
ATP-mediated potassium recycling in the cochlear supporting cellsYan Zhu, Hong-Bo ZhaoBiochemical and Biophysical Research Communications|September 18, 2012
ATP activates P2X receptors to mediate gap junctional coupling in the cochleaYan Zhu, Hong-Bo ZhaoPflugers Archiv : European Journal of Physiology|May 21, 2008
ATP activates P2x receptors and requires extracellular Ca(++) participation to modify outer hair cell nonlinear capacitanceNing Yu, Hong-Bo ZhaoPlos One|November 26, 2009
Modulation of outer hair cell electromotility by cochlear supporting cells and gap junctionsNing Yu, Hong-Bo ZhaoCell Communication & Adhesion|November 4, 2008
Hemichannel-mediated inositol 1,4,5-trisphosphate (IP3) release in the cochlea: a novel mechanism of IP3 intercellular signalingDavid G Gossman, Hong-Bo ZhaoPflugers Archiv : European Journal of Physiology|June 5, 2003
Excitation of fluorescent dyes inactivates the outer hair cell integral membrane motor protein prestin and betrays its lateral mobilityJoseph Santos-Sacchi, Hong-Bo ZhaoPageof 9