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Phytomedicine : International Journal of Phytotherapy and Phytopharmacology|December 9, 2025
Polygonum cuspidatum alleviates hyperuricemia-induced renal injury via rectifying amino acid metabolism disordersYuqing Ma, Lei Peng, Honghao Song, et al.
BMC Cancer|November 28, 2020
Risk factors and socio-economic burden in pancreatic ductal adenocarcinoma operation: a machine learning based analysisYijue Zhang, Sibo Zhu, Zhiqing Yuan, et al.
Journal of Hepatology|May 28, 2014
Inhibition of glycogen synthase kinase 3 ameliorates liver ischemia/reperfusion injury via an energy-dependent mitochondrial mechanismHailong Fu, Haitao Xu, Huan Chen, et al.
The Journal of Biological Chemistry|December 3, 2005
Different residues mediate recognition of 1-O-oleyllysophosphatidic acid and rosiglitazone in the ligand binding domain of peroxisome proliferator-activated receptor gammaTamotsu Tsukahara, Ryoko Tsukahara, Satoshi Yasuda, et al.
Journal of Cellular and Molecular Medicine|October 27, 2020
Up-regulation of FoxO1 contributes to adverse vascular remodelling in type 1 diabetic ratsJingjin Liu, Xiang Xie, Dan Yan, et al.
Molecular Immunology|April 29, 2020
TAOK1 positively regulates TLR4-induced inflammatory responses by promoting ERK1/2 activation in macrophagesLingxi Zhu, Qingzhuo Yu, Peng Gao, et al.
Free Radical Biology & Medicine|March 9, 2026
STAT3 Inhibition Ameliorates Cognitive Dysfunction in Type 2 Diabetes Mellitus by Modulating Astrocytes via Nrf2 Pathway ActivationJue Wang, Peixian Chen, Yonghua Li, et al.
Bioorganic & Medicinal Chemistry|March 14, 2009
Discovery of 2-(5-nitrothiazol-2-ylthio)benzo[d]thiazoles as novel c-Jun N-terminal kinase inhibitorsSurya K De, Li-Hsing Chen, John L Stebbins, et al.
Journal of Medicinal Chemistry|March 11, 2009
Design, synthesis, and structure-activity relationship of substrate competitive, selective, and in vivo active triazole and thiadiazole inhibitors of the c-Jun N-terminal kinaseSurya K De, John L Stebbins, Li-Hsing Chen, et al.
International Immunopharmacology|May 11, 2026
PRC1 impairs sepsis host defense by suppressing NF-κB/JAK-STAT signaling and macrophage glycolytic reprogrammingHuawei Wei, Xiaoyan Meng, Haoyu Fan, et al.
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