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Saudi Medical Journal|May 14, 2010
High mobility group box-1 and cardiovascular diseasesHua-Sheng Ding, Jun YangCardiovascular Toxicology|October 21, 2021
AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic ModelsPeng Zhong, Jianye Peng, Tao Liu, et al.International Journal of Molecular Medicine|February 27, 2013
High mobility group box 1 and kidney diseases (Review)Ping Zhu, Li Xie, Hua-Sheng Ding, et al.Molecular Biology Reports|February 15, 2012
Interleukin-17 contributes to cardiovascular diseasesHua-Sheng Ding, Jun Yang, Jian Yang, et al.Journal of Inflammation Research|May 22, 2024
Analyzing Immune Cell Infiltration and Copper Metabolism in Diabetic Foot UlcersWen-Juan Yi, Yifan Yuan, Qionglin Bao, et al.Oncotarget|May 19, 2018
MiR-33 promotes myocardial fibrosis by inhibiting MMP16 and stimulating p38 MAPK signalingZhen Chen, Hua-Sheng Ding, Xin Guo, et al.Gene|June 4, 2013
The HMGB1-TLR4 axis contributes to myocardial ischemia/reperfusion injury via regulation of cardiomyocyte apoptosisHua-Sheng Ding, Jun Yang, Ping Chen, et al.Biochemical and Biophysical Research Communications|March 15, 2018
Regulator of G-protein signalling 5 deficiency impairs ventricular remodelling after myocardial infarction by promoting NF-κB and MAPK signalling in miceHua-Sheng Ding, Yan Huang, Zhen Chen, et al.Gene|August 15, 2012
High mobility group [corrected] box 1 mediates neutrophil recruitment in myocardial ischemia-reperfusion injury through toll like receptor 4-related pathwayHua-Sheng Ding, Jun Yang, Fei-Li Gong, et al.International Journal of Molecular Medicine|July 20, 2016
Radioprotective 105 kDa protein attenuates ischemia/reperfusion-induced myocardial apoptosis and autophagy by inhibiting the activation of the TLR4/NF-κB signaling pathway in ratsXin Guo, Hong Jiang, Jun Yang, et al.Pageof 2