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Molecular and Cellular Biology|October 23, 2002
Activation of Akt/protein kinase B overcomes a G(2)/m cell cycle checkpoint induced by DNA damageEugene S Kandel, Jennifer Skeen, Nathan Majewski, et al.Cancer Research|August 3, 2007
Identification of S664 TSC2 phosphorylation as a marker for extracellular signal-regulated kinase mediated mTOR activation in tuberous sclerosis and human cancerLi Ma, Julie Teruya-Feldstein, Pauline Bonner, et al.Oncogene|November 20, 2002
Rabies virus P and small P products interact directly with PML and reorganize PML nuclear bodiesDanielle Blondel, Tarick Regad, Nicolas Poisson, et al.Nature Communications|November 19, 2021
Genetic fusions favor tumorigenesis through degron loss in oncogenesJing Liu, Collin Tokheim, Jonathan D Lee, et al.Blood|January 24, 2008
Npm1 is a haploinsufficient suppressor of myeloid and lymphoid malignancies in the mousePaolo Sportoletti, Silvia Grisendi, Samia M Majid, et al.Physical Chemistry Chemical Physics : PCCP|January 25, 2017
Comment on "Magnetic-field-enabled resolution enhancement in super-resolution imaging" by M. Zhang et al., Phys. Chem. Chem. Phys., 2015, 17, 6722-6727Stephan Bergmann, Viola Mönkemöller, Thomas HuserJournal of Immunology (Baltimore, Md. : 1950)|November 16, 2010
CD8 T cell-intrinsic GITR is required for T cell clonal expansion and mouse survival following severe influenza infectionLaura M Snell, Ann J McPherson, Gloria H Y Lin, et al.The Journal of Experimental Medicine|December 22, 2004
Role of Dok-1 and Dok-2 in leukemia suppressionMasaru Niki, Antonio Di Cristofano, Mingming Zhao, et al.Molecular and Cellular Biology|March 12, 2014
The tumor suppressor PML specifically accumulates at RPA/Rad51-containing DNA damage repair foci but is nonessential for DNA damage-induced fibroblast senescenceSandra Münch, Stefanie Weidtkamp-Peters, Karolin Klement, et al.The Journal of Biological Chemistry|July 13, 2006
Promyelocytic leukemia activates Chk2 by mediating Chk2 autophosphorylationShutong Yang, Jae-Hoon Jeong, Alexandra L Brown, et al.Pageof 50