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Brain, Behavior, and Immunity|August 8, 2017
Electroacupuncture inhibits NLRP3 inflammasome activation through CB2 receptors in inflammatory painFang Gao, Hong-Chun Xiang, Hong-Ping Li, et al.
Cell Reports|May 11, 2016
Chloride Homeostasis Critically Regulates Synaptic NMDA Receptor Activity in Neuropathic PainLingyong Li, Shao-Rui Chen, Hong Chen, et al.
The Journal of Pain|July 15, 2010
Electroacupuncture increases CB2 receptor expression on keratinocytes and infiltrating inflammatory cells in inflamed skin tissues of ratsJing Zhang, Lin Chen, Tangfeng Su, et al.
Stroke|October 26, 2018
Focal Cerebral Ischemia and Reperfusion Induce Brain Injury Through α2δ-1-Bound NMDA ReceptorsYi Luo, Huijie Ma, Jing-Jing Zhou, et al.
The Journal of Clinical Investigation|October 23, 2025
Spinal α2δ-1 induces GluA3 degradation to regulate assembly of calcium-permeable AMPA receptors and pain hypersensitivityMeng-Hua Zhou, Shao-Rui Chen, Daozhong Jin, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 18, 2022
HDAC2 in Primary Sensory Neurons Constitutively Restrains Chronic Pain by Repressing α2δ-1 Expression and Associated NMDA Receptor ActivityJixiang Zhang, Shao-Rui Chen, Meng-Hua Zhou, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 8, 2018
Nerve Injury-Induced Chronic Pain Is Associated with Persistent DNA Methylation Reprogramming in Dorsal Root GanglionJudit Garriga, Geoffroy Laumet, Shao-Rui Chen, et al.
The Journal of Biological Chemistry|December 22, 2011
Up-regulation of Cavβ3 subunit in primary sensory neurons increases voltage-activated Ca2+ channel activity and nociceptive input in neuropathic painLi Li, Xue-Hong Cao, Shao-Rui Chen, et al.
The Journal of Pain|May 5, 2009
Endogenous anandamide and cannabinoid receptor-2 contribute to electroacupuncture analgesia in ratsLin Chen, Jing Zhang, Fan Li, et al.
Science Translational Medicine|October 22, 2025
LRRC8A constitutively inhibits pain hypersensitivity in rodent models by restraining NMDA receptor activity at spinal cord synapsesMeichun Deng, Shao-Rui Chen, Meng-Hua Zhou, et al.
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