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Plos Pathogens|October 8, 2011
Evaluating the sensitivity of Mycobacterium tuberculosis to biotin deprivation using regulated gene expressionSae Woong Park, Marcus Klotzsche, Daniel J Wilson, et al.Mbio|June 23, 2023
Metabolically distinct roles of NAD synthetase and NAD kinase define the essentiality of NAD and NADP in <i>Mycobacterium tuberculosis</i>Ritu Sharma, Travis E Hartman, Tiago Beites, et al.Proceedings of the National Academy of Sciences of the United States of America|March 28, 2020
Oncogenic human herpesvirus hijacks proline metabolism for tumorigenesisUn Yung Choi, Jae Jin Lee, Angela Park, et al.ACS Infectious Diseases|March 14, 2024
Targeting <i>Mycobacterium tuberculosis</i> Persistence through Inhibition of the Trehalose Catalytic ShiftKarishma Kalera, Rachel Liu, Juhyeon Lim, et al.BMC Microbiology|December 31, 2010
Regulation of polar peptidoglycan biosynthesis by Wag31 phosphorylation in mycobacteriaCharul Jani, Hyungjin Eoh, Jae Jin Lee, et al.Experimental & Molecular Medicine|February 27, 2024
NOTCH localizes to mitochondria through the TBC1D15-FIS1 interaction and is stabilized via blockade of E3 ligase and CDK8 recruitment to reprogram tumor-initiating cellsHye Yeon Choi, Yicheng Zhu, Xuyao Zhao, et al.Plos One|February 8, 2024
TXNIP-mediated crosstalk between oxidative stress and glucose metabolismStephanie Kim, Jianning Ge, Dokyun Kim, et al.Research Square|September 24, 2024
Trehalose catalytic shift is an intrinsic factor in Mycobacterium tuberculosis that enhances phenotypic heterogeneity and multidrug resistanceHyungjin Eoh, Jae Jin Lee, Daniel Swanson, et al.Nature Microbiology|June 10, 2020
A nutrient-limited screen unmasks rifabutin hyperactivity for extensively drug-resistant Acinetobacter baumanniiBrian Luna, Vincent Trebosc, Bosul Lee, et al.Nature Communications|July 11, 2025
Trehalose catalytic shift inherently enhances phenotypic heterogeneity and multidrug resistance in Mycobacterium tuberculosisJae Jin Lee, Daniel H Swanson, Sun-Kyung Lee, et al.Pageof 6