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Neurobiology of Disease|April 22, 2009
GSK3beta, a centre-staged kinase in neuropsychiatric disorders, modulates long term memory by inhibitory phosphorylation at serine-9I Dewachter, L Ris, T Jaworski, et al.Nature Communications|August 8, 2019
Molecular understanding of label-free second harmonic imaging of microtubulesV Van Steenbergen, W Boesmans, Z Li, et al.British Journal of Pharmacology|February 28, 2007
5-HT4 receptor agonists increase sAPPalpha levels in the cortex and hippocampus of male C57BL/6j miceM Cachard-Chastel, F Lezoualc'h, I Dewachter, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 31, 2000
Aging increased amyloid peptide and caused amyloid plaques in brain of old APP/V717I transgenic mice by a different mechanism than mutant presenilin1I Dewachter, J Van Dorpe, L Smeijers, et al.The Journal of Biological Chemistry|March 30, 2001
Mutant presenilins disturb neuronal calcium homeostasis in the brain of transgenic mice, decreasing the threshold for excitotoxicity and facilitating long-term potentiationI Schneider, D Reverse, I Dewachter, et al.Neurobiology of Disease|February 15, 2000
Behavioral disturbances without amyloid deposits in mice overexpressing human amyloid precursor protein with Flemish (A692G) or Dutch (E693Q) mutationS Kumar-Singh, I Dewachter, D Moechars, et al.The Journal of Biological Chemistry|February 26, 1999
Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brainD Moechars, I Dewachter, K Lorent, et al.Neurobiology of Aging|August 4, 2007
Deregulation of NMDA-receptor function and down-stream signaling in APP[V717I] transgenic miceI Dewachter, R K Filipkowski, C Priller, et al.The American Journal of Pathology|October 6, 2000
Prominent cerebral amyloid angiopathy in transgenic mice overexpressing the london mutant of human APP in neuronsJ Van Dorpe, L Smeijers, I Dewachter, et al.Molecular Psychiatry|February 13, 2013
Increased expression of BIN1 mediates Alzheimer genetic risk by modulating tau pathologyJ Chapuis, F Hansmannel, M Gistelinck, et al.Pageof 2