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Critical Care Medicine|August 1, 1997
Apoptosis in lymphoid and parenchymal cells during sepsis: findings in normal and T- and B-cell-deficient miceR S Hotchkiss, P E Swanson, J P Cobb, et al.Shock (Augusta, Ga.)|October 26, 2000
Injection of iron compounds followed by induction of the stress response causes tissue injury and apoptosisA K Jacob, R S Hotchkiss, P E Swanson, et al.Shock (Augusta, Ga.)|August 4, 1998
Pyrrolidine dithiocarbamate activates the heat shock response and thereby induces apoptosis in primed endothelial cellsS L DeMeester, T G Buchman, Y Qiu, et al.Critical Care Medicine|September 29, 1998
Nitric oxide inhibits stress-induced endothelial cell apoptosisS L DeMeester, Y Qiu, T G Buchman, et al.Surgery|August 26, 1998
Stress-induced fractal rearrangement of the endothelial cell cytoskeleton causes apoptosisS L DeMeester, J P Cobb, R S Hotchkiss, et al.Surgery|August 24, 1999
Inducible nitric oxide synthase (iNOS) gene deficiency increases the mortality of sepsis in miceJ P Cobb, R S Hotchkiss, P E Swanson, et al.Surgery|August 1, 1997
Endothelial cell apoptosis is accelerated by inorganic iron and heat via an oxygen radical dependent mechanismA K Jacob, R S Hotchkiss, S L DeMeester, et al.Proceedings of the National Academy of Sciences of the United States of America|December 10, 1999
Prevention of lymphocyte cell death in sepsis improves survival in miceR S Hotchkiss, K W Tinsley, P E Swanson, et al.Shock (Augusta, Ga.)|January 19, 2000
Caspases -2, -3, -6, and -9, but not caspase-1, are activated in sepsis-induced thymocyte apoptosisK W Tinsley, S L Cheng, T G Buchman, et al.Critical Care Medicine|July 13, 2000
Role of CuZn superoxide dismutase in regulating lymphocyte apoptosis during sepsisB D Freeman, A G Reaume, P E Swanson, et al.Pageof 7