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The American Journal of Physiology|January 1, 1991
Sepsis does not impair tricarboxylic acid cycle in the heartR S Hotchkiss, S K Song, J J Neil, et al.Journal of Immunology (Baltimore, Md. : 1950)|March 22, 2000
p53-dependent and -independent pathways of apoptotic cell death in sepsisR S Hotchkiss, K W Tinsley, J J Hui, et al.The American Journal of Physiology|October 1, 1991
Evaluation of the role of cellular hypoxia in sepsis by the hypoxic marker [18F]fluoromisonidazoleR S Hotchkiss, R S Rust, C S Dence, et al.Critical Care Medicine|August 14, 1999
Apoptotic cell death in patients with sepsis, shock, and multiple organ dysfunctionR S Hotchkiss, P E Swanson, B D Freeman, et al.Critical Care Medicine|September 29, 2000
Rapid onset of intestinal epithelial and lymphocyte apoptotic cell death in patients with trauma and shockR S Hotchkiss, R E Schmieg, P E Swanson, et al.Journal of Immunology (Baltimore, Md. : 1950)|April 14, 1999
Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsisR S Hotchkiss, P E Swanson, C M Knudson, et al.Shock (Augusta, Ga.)|March 10, 2001
Injury in the era of genomicsJ P Cobb, B H Brownstein, M A Watson, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 22, 2001
Sepsis-induced apoptosis causes progressive profound depletion of B and CD4+ T lymphocytes in humansR S Hotchkiss, K W Tinsley, P E Swanson, et al.Nature Immunology|March 23, 2001
Caspase inhibitors improve survival in sepsis: a critical role of the lymphocyteR S Hotchkiss, K C Chang, P E Swanson, et al.Pageof 7