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The EMBO Journal|February 1, 1989
Galanin activates nucleotide-dependent K+ channels in insulin-secreting cells via a pertussis toxin-sensitive G-proteinM J Dunne, M J Bullett, G D Li, et al.Biochemical Society Transactions|October 25, 2007
Glucokinase activators: molecular tools for studying the physiology of insulin-secreting cellsD Johnson, R M Shepherd, D Gill, et al.Archives of Disease in Childhood|June 12, 1998
Intra-arterial calcium stimulation test in the investigation of hyperinsulinaemic hypoglycaemiaL J Abernethy, D C Davidson, G L Lamont, et al.Diabetic Medicine : a Journal of the British Diabetic Association|September 25, 2018
Therapies and outcomes of congenital hyperinsulinism-induced hypoglycaemiaI Banerjee, M Salomon-Estebanez, P Shah, et al.The Journal of Membrane Biology|September 1, 1988
The gating of nucleotide-sensitive K+ channels in insulin-secreting cells can be modulated by changes in the ratio ATP4-/ADP3- and by nonhydrolyzable derivatives of both ATP and ADPM J Dunne, J A West-Jordan, R J Abraham, et al.The EMBO Journal|August 1, 1988
Activators of protein kinase C depolarize insulin-secreting cells by closing K+ channelsC B Wollheim, M J Dunne, B Peter-Riesch, et al.Journal of Medical Genetics|March 21, 1998
Prenatal detection of trisomy 21 and 18 from amniotic fluid by quantitative fluorescent polymerase chain reactionT Tóth, I Findlay, C Papp, et al.Prenatal Diagnosis|August 26, 1998
Prenatal detection of trisomy 13 from amniotic fluid by quantitative fluorescent polymerase chain reactionT Tóth, I Findlay, C Papp, et al.Diabetes|June 27, 2000
Glucose modulation of insulin mRNA levels is dependent on transcription factor PDX-1 and occurs independently of changes in intracellular Ca2+W M Macfarlane, R M Shepherd, K E Cosgrove, et al.Clinical Endocrinology|January 26, 2013
Integrating genetic and imaging investigations into the clinical management of congenital hyperinsulinismI Banerjee, B Avatapalle, R Padidela, et al.Pageof 15