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I Fuse

Showing results (11-20 of 67) with videos related to

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Biochemical and Biophysical Research Communications|July 31, 1987
Stimulations of arachidonate release and inositol-1,4,5-triphosphate formation are mediated by distinct G-proteins in human plateletsI Fuse, H H Tai
Seminars in Thrombosis and Hemostasis|May 11, 2000
Pathogenesis of a bleeding disorder characterized by platelet unresponsiveness to thromboxane A2I Fuse, W Higuchi, Y Aizawa
Nihon Ketsueki Gakkai Zasshi : Journal of Japan Haematological Society|December 1, 1989
Characterization of platelet cytoplasmic Ca2+ mobilization in patients with congenital cyclo-oxygenase deficiency and with defective platelet aggregation to A23187I Fuse, A Hattori, R Nagayama
Acta Haematologica|January 13, 2001
Arg60 Leu mutation in the first cytoplasmic loop of the platelet thromboxane A2 receptor is not essential for mediating inhibitory coupling between the receptor and adenylyl cyclaseI Fuse, W Higuchi, Y Aizawa
Rinsho Byori. the Japanese Journal of Clinical Pathology|March 1, 1992
[Analysis of platelet shape change, inositol metabolism, and Ca mobilization in patients with platelet dysfunction]I Fuse, A Hattori, W Higuchi
The Journal of Biological Chemistry|March 5, 1989
Phorbol ester, 1,2-diacylglycerol, and collagen induce inhibition of arachidonic acid incorporation into phospholipids in human plateletsI Fuse, T Iwanaga, H H Tai
Nihon Rinsho. Japanese Journal of Clinical Medicine|February 1, 1992
[PI metabolism and Ca mobilization in patients with platelet dysfunction]I Fuse, A Hattori, W Higuchi, et al.
Platelets|June 28, 2006
Inhibitory mechanism of human platelet aggregation by nafamostat mesilateI Fuse, W Higuchi, K Toba, et al.
Thrombosis and Haemostasis|December 14, 1999
Mutations of the platelet thromboxane A2 (TXA2) receptor in patients characterized by the absence of TXA2-induced platelet aggregation despite normal TXA2 binding activityW Higuchi, I Fuse, A Hattori, et al.
Japanese Heart Journal|January 1, 1996
Abnormal function of platelet G proteins in long QT syndromeT Washizuka, Y Aizawa, I Fuse, et al.
Pageof 7

Showing results (11-20 of 67) with videos related to

Sort By:
Pageof 7
Biochemical and Biophysical Research Communications|July 31, 1987
Stimulations of arachidonate release and inositol-1,4,5-triphosphate formation are mediated by distinct G-proteins in human plateletsI Fuse, H H Tai
Seminars in Thrombosis and Hemostasis|May 11, 2000
Pathogenesis of a bleeding disorder characterized by platelet unresponsiveness to thromboxane A2I Fuse, W Higuchi, Y Aizawa
Nihon Ketsueki Gakkai Zasshi : Journal of Japan Haematological Society|December 1, 1989
Characterization of platelet cytoplasmic Ca2+ mobilization in patients with congenital cyclo-oxygenase deficiency and with defective platelet aggregation to A23187I Fuse, A Hattori, R Nagayama
Acta Haematologica|January 13, 2001
Arg60 Leu mutation in the first cytoplasmic loop of the platelet thromboxane A2 receptor is not essential for mediating inhibitory coupling between the receptor and adenylyl cyclaseI Fuse, W Higuchi, Y Aizawa
Rinsho Byori. the Japanese Journal of Clinical Pathology|March 1, 1992
[Analysis of platelet shape change, inositol metabolism, and Ca mobilization in patients with platelet dysfunction]I Fuse, A Hattori, W Higuchi
The Journal of Biological Chemistry|March 5, 1989
Phorbol ester, 1,2-diacylglycerol, and collagen induce inhibition of arachidonic acid incorporation into phospholipids in human plateletsI Fuse, T Iwanaga, H H Tai
Nihon Rinsho. Japanese Journal of Clinical Medicine|February 1, 1992
[PI metabolism and Ca mobilization in patients with platelet dysfunction]I Fuse, A Hattori, W Higuchi, et al.
Platelets|June 28, 2006
Inhibitory mechanism of human platelet aggregation by nafamostat mesilateI Fuse, W Higuchi, K Toba, et al.
Thrombosis and Haemostasis|December 14, 1999
Mutations of the platelet thromboxane A2 (TXA2) receptor in patients characterized by the absence of TXA2-induced platelet aggregation despite normal TXA2 binding activityW Higuchi, I Fuse, A Hattori, et al.
Japanese Heart Journal|January 1, 1996
Abnormal function of platelet G proteins in long QT syndromeT Washizuka, Y Aizawa, I Fuse, et al.
Pageof 7