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I H Chaudry

Showing results (51-60 of 400) with videos related to

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The American Journal of Physiology|April 1, 1995
ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitationP Wang, Z F Ba, I H Chaudry
The Journal of Surgical Research|August 12, 1998
Does hepatocellular injury in sepsis involve apoptosis?A Ayala, T A Evans, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|February 1, 1991
Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac outputP Wang, Z F Ba, I H Chaudry
Biochimica Et Biophysica Acta|November 21, 1997
Administration of ATP-MgCl2 following hemorrhage and resuscitation increases hepatic phosphoenolpyruvate carboxykinase and decreases pyruvate kinase activitiesM S Mahmoud, P Wang, I H Chaudry
The American Journal of Physiology|November 14, 1997
Male sex steroids are responsible for depressing macrophage immune function after trauma-hemorrhageM W Wichmann, A Ayala, I H Chaudry
The American Journal of Physiology|December 1, 1992
Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitationP Wang, Z F Ba, I H Chaudry
Advances in Shock Research|January 1, 1979
Impairment of reticuloendothelial system function with sepsis and its improvement with ATP-MgCl2 plus glucose administrationI H Chaudry, H Hirasawa, A E Baue
The American Journal of Physiology|August 1, 1993
Mechanism of diltiazem's immunomodulatory effects after hemorrhage and resuscitationD R Meldrum, A Ayala, I H Chaudry
The American Journal of Physiology|May 1, 1993
ATP-MgCl2 restores gut absorptive capacity early after trauma-hemorrhagic shockG Singh, K I Chaudry, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|April 1, 1997
Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6P Wang, Z F Ba, I H Chaudry
Pageof 40

Showing results (51-60 of 400) with videos related to

Sort By:
Pageof 40
The American Journal of Physiology|April 1, 1995
ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitationP Wang, Z F Ba, I H Chaudry
The Journal of Surgical Research|August 12, 1998
Does hepatocellular injury in sepsis involve apoptosis?A Ayala, T A Evans, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|February 1, 1991
Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac outputP Wang, Z F Ba, I H Chaudry
Biochimica Et Biophysica Acta|November 21, 1997
Administration of ATP-MgCl2 following hemorrhage and resuscitation increases hepatic phosphoenolpyruvate carboxykinase and decreases pyruvate kinase activitiesM S Mahmoud, P Wang, I H Chaudry
The American Journal of Physiology|November 14, 1997
Male sex steroids are responsible for depressing macrophage immune function after trauma-hemorrhageM W Wichmann, A Ayala, I H Chaudry
The American Journal of Physiology|December 1, 1992
Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitationP Wang, Z F Ba, I H Chaudry
Advances in Shock Research|January 1, 1979
Impairment of reticuloendothelial system function with sepsis and its improvement with ATP-MgCl2 plus glucose administrationI H Chaudry, H Hirasawa, A E Baue
The American Journal of Physiology|August 1, 1993
Mechanism of diltiazem's immunomodulatory effects after hemorrhage and resuscitationD R Meldrum, A Ayala, I H Chaudry
The American Journal of Physiology|May 1, 1993
ATP-MgCl2 restores gut absorptive capacity early after trauma-hemorrhagic shockG Singh, K I Chaudry, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|April 1, 1997
Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6P Wang, Z F Ba, I H Chaudry
Pageof 40