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Acta Neuropathologica Communications|July 6, 2022
Terminal complement pathway activation drives synaptic loss in Alzheimer's disease modelsSarah M Carpanini, Megan Torvell, Ryan J Bevan, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 2, 2022
Analysis of Complement Gene Expression, Clinical Associations, and Biodistribution of Complement Proteins in the Synovium of Early Rheumatoid Arthritis Patients Reveals Unique Pathophysiologic FeaturesNirmal K Banda, Kevin D Deane, Elizabeth A Bemis, et al.Frontiers in Neurology|March 23, 2017
Microbial Neuraminidase Induces a Moderate and Transient Myelin Vacuolation Independent of Complement System ActivationPablo Granados-Durán, María Dolores López-Ávalos, Manuel Cifuentes, et al.Clinical Immunology (Orlando, Fla.)|June 5, 2017
Hexamerization-enhanced CD20 antibody mediates complement-dependent cytotoxicity in serum genetically deficient in C9Ronald P Taylor, Margaret A Lindorfer, Erika M Cook, et al.Plos Genetics|January 27, 2025
Wild-type bone marrow cells repopulate tissue resident macrophages and reverse the impacts of homozygous CSF1R mutationDylan Carter-Cusack, Stephen Huang, Sahar Keshvari, et al.The American Journal of Pathology|August 7, 2007
Myasthenia gravis thymus: complement vulnerability of epithelial and myoid cells, complement attack on them, and correlations with autoantibody statusMaria I Leite, Margaret Jones, Philipp Ströbel, et al.Med (New York, N.Y.)|February 15, 2024
Complement dysregulation is a prevalent and therapeutically amenable feature of long COVIDKirsten Baillie, Helen E Davies, Samuel B K Keat, et al.ACS Energy Letters|April 18, 2024
Cation Effects on the Acidic Oxygen Reduction Reaction at Carbon SurfacesJ L Hübner, L E B Lucchetti, H N Nong, et al.Journal of Immunological Methods|February 5, 2013
Distribution and determinants of circulating complement factor H concentration determined by a high-throughput immunonephelometric assayReecha Sofat, P Patrizia Mangione, J Ruth Gallimore, et al.Journal of Immunology (Baltimore, Md. : 1950)|February 9, 2005
Decay-accelerating factor (CD55) is expressed by neurons in response to chronic but not acute autoimmune central nervous system inflammation associated with complement activationJohan van Beek, Marjan van Meurs, Bert A 't Hart, et al.Pageof 72