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I L Grupp

Showing results (51-60 of 60) with videos related to

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The Journal of Clinical Investigation|October 15, 1996
Ablation of the murine alpha myosin heavy chain gene leads to dosage effects and functional deficits in the heartW K Jones, I L Grupp, T Doetschman, et al.
The Journal of Biological Chemistry|July 27, 1999
Cardiac-specific overexpression of the alpha(1) subunit of the L-type voltage-dependent Ca(2+) channel in transgenic mice. Loss of isoproterenol-induced contractionJ N Muth, H Yamaguchi, G Mikala, et al.
Circulation Research|October 31, 1998
Enhanced myocardial contractility and increased Ca2+ transport function in transgenic hearts expressing the fast-twitch skeletal muscle sarcoplasmic reticulum Ca2+-ATPaseE Loukianov, Y Ji, I L Grupp, et al.
Circulation Research|July 13, 1999
Mouse model of a familial hypertrophic cardiomyopathy mutation in alpha-tropomyosin manifests cardiac dysfunctionM Muthuchamy, K Pieples, P Rethinasamy, et al.
The Journal of Pharmacology and Experimental Therapeutics|June 1, 1996
Positive inotropic effects of the calcium sensitizer CGP 48506 in failing human myocardiumJ Neumann, T Eschenhagen, I L Grupp, et al.
Circulation Research|December 16, 1998
Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse heartsD L Baker, K Hashimoto, I L Grupp, et al.
American Journal of Physiology. Heart and Circulatory Physiology|December 21, 2000
Progression from hypertrophic to dilated cardiomyopathy in mice that express a mutant myosin transgeneK Freeman, C Colon-Rivera, M C Olsson, et al.
Circulation Research|May 1, 1996
Phospholamban gene dosage effects in the mammalian heartW Luo, B M Wolska, I L Grupp, et al.
Journal of Molecular and Cellular Cardiology|May 10, 2001
The enhanced contractility of the phospholamban-deficient mouse heart persists with agingJ P Slack, I L Grupp, R Dash, et al.
Circulation Research|December 1, 1996
Compensatory mechanisms associated with the hyperdynamic function of phospholamban-deficient mouse heartsG Chu, W Luo, J P Slack, et al.
Pageof 6

Showing results (51-60 of 60) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 60 results.
The Journal of Clinical Investigation|October 15, 1996
Ablation of the murine alpha myosin heavy chain gene leads to dosage effects and functional deficits in the heartW K Jones, I L Grupp, T Doetschman, et al.
The Journal of Biological Chemistry|July 27, 1999
Cardiac-specific overexpression of the alpha(1) subunit of the L-type voltage-dependent Ca(2+) channel in transgenic mice. Loss of isoproterenol-induced contractionJ N Muth, H Yamaguchi, G Mikala, et al.
Circulation Research|October 31, 1998
Enhanced myocardial contractility and increased Ca2+ transport function in transgenic hearts expressing the fast-twitch skeletal muscle sarcoplasmic reticulum Ca2+-ATPaseE Loukianov, Y Ji, I L Grupp, et al.
Circulation Research|July 13, 1999
Mouse model of a familial hypertrophic cardiomyopathy mutation in alpha-tropomyosin manifests cardiac dysfunctionM Muthuchamy, K Pieples, P Rethinasamy, et al.
The Journal of Pharmacology and Experimental Therapeutics|June 1, 1996
Positive inotropic effects of the calcium sensitizer CGP 48506 in failing human myocardiumJ Neumann, T Eschenhagen, I L Grupp, et al.
Circulation Research|December 16, 1998
Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse heartsD L Baker, K Hashimoto, I L Grupp, et al.
American Journal of Physiology. Heart and Circulatory Physiology|December 21, 2000
Progression from hypertrophic to dilated cardiomyopathy in mice that express a mutant myosin transgeneK Freeman, C Colon-Rivera, M C Olsson, et al.
Circulation Research|May 1, 1996
Phospholamban gene dosage effects in the mammalian heartW Luo, B M Wolska, I L Grupp, et al.
Journal of Molecular and Cellular Cardiology|May 10, 2001
The enhanced contractility of the phospholamban-deficient mouse heart persists with agingJ P Slack, I L Grupp, R Dash, et al.
Circulation Research|December 1, 1996
Compensatory mechanisms associated with the hyperdynamic function of phospholamban-deficient mouse heartsG Chu, W Luo, J P Slack, et al.
Pageof 6