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Proceedings of the National Academy of Sciences of the United States of America|August 2, 2001
SMAD4 mutations in colorectal cancer probably occur before chromosomal instability, but after divergence of the microsatellite instability pathwayK L Woodford-Richens, A J Rowan, P Gorman, et al.Princess Takamatsu Symposia|January 1, 1989
Genetic analysis of colorectal cancerW F Bodmer, S Cottrell, A M Frischauf, et al.Diseases of the Colon and Rectum|May 27, 1999
Distribution of carcinoembryonic antigen and biologic behavior in colorectal carcinomaJ C Kim, M S Han, H K Lee, et al.Neurology|June 16, 2005
New onset geriatric epilepsy: a randomized study of gabapentin, lamotrigine, and carbamazepineA J Rowan, R E Ramsay, J F Collins, et al.The New England Journal of Medicine|September 17, 1998
A comparison of four treatments for generalized convulsive status epilepticus. Veterans Affairs Status Epilepticus Cooperative Study GroupD M Treiman, P D Meyers, N Y Walton, et al.Gut|June 3, 2008
Inherited predisposition to colorectal adenomas caused by multiple rare alleles of MUTYH but not OGG1, NUDT1, NTH1 or NEIL 1, 2 or 3A R Dallosso, S Dolwani, N Jones, et al.Nature Communications|December 19, 2025
The molecular cartography of malignant and benign sebaceous tumoursI Ferreira, O M Rueda, L van der Weyden, et al.Gut|February 8, 2006
Disease severity and genetic pathways in attenuated familial adenomatous polyposis vary greatly but depend on the site of the germline mutationO M Sieber, S Segditsas, A L Knudsen, et al.Proceedings of the National Academy of Sciences of the United States of America|February 28, 2002
Whole-gene APC deletions cause classical familial adenomatous polyposis, but not attenuated polyposis or "multiple" colorectal adenomasO M Sieber, H Lamlum, M D Crabtree, et al.Pageof 8