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Nature|November 11, 1993
Complementation by the protein tyrosine kinase JAK2 of a mutant cell line defective in the interferon-gamma signal transduction pathwayD Watling, D Guschin, M Müller, et al.Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|January 30, 1995
Regulation and mechanisms of gene amplificationK A Smith, M L Agarwal, M V Chernov, et al.Proceedings of the National Academy of Sciences of the United States of America|September 17, 1996
Use of genetic suppressor elements to dissect distinct biological effects of separate p53 domainsV S Ossovskaya, I A Mazo, M V Chernov, et al.The EMBO Journal|October 16, 1995
Mutant U5A cells are complemented by an interferon-alpha beta receptor subunit generated by alternative processing of a new member of a cytokine receptor gene clusterG Lutfalla, S J Holland, E Cinato, et al.Immunity|November 1, 1994
Molecular analysis of G1B and G3A IFN gamma mutants reveals that defects in CIITA or RFX result in defective class II MHC and Ii gene inductionK C Chin, C Mao, C Skinner, et al.Journal of Virology|October 1, 1983
Persistent infection of some standard cell lines by lymphocytic choriomeningitis virus: transmission of infection by an intracellular agentB A van der Zeijst, B E Noyes, M E Mirault, et al.Oncogene|June 23, 2001
Regulation of p53 expression by the RAS-MAP kinase pathwayM L Agarwal, C V Ramana, M Hamilton, et al.The EMBO Journal|March 17, 1997
Transgenic mice with p53-responsive lacZ: p53 activity varies dramatically during normal development and determines radiation and drug sensitivity in vivoE A Komarova, M V Chernov, R Franks, et al.EMBO Reports|July 21, 2001
CARD4/Nod1 mediates NF-kappaB and JNK activation by invasive Shigella flexneriS E Girardin, R Tournebize, M Mavris, et al.Proceedings of the National Academy of Sciences of the United States of America|March 4, 1997
Multiple mechanisms of N-phosphonacetyl-L-aspartate resistance in human cell lines: carbamyl-P synthetase/aspartate transcarbamylase/dihydro-orotase gene amplification is frequent only when chromosome 2 is rearrangedK A Smith, O B Chernova, R P Groves, et al.Pageof 22