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Annals of Human Genetics|October 19, 2006
Epistasis between type 2 diabetes susceptibility Loci on chromosomes 1q21-25 and 10q23-26 in northern EuropeansS Wiltshire, J T Bell, C J Groves, et al.
Diabetologia|June 6, 2008
Diabetes mellitus among young adults in Sri Lanka--role of GAD antibodies in classification and treatment: the Sri Lanka Young Diabetes studyP Katulanda, B Shine, G W Katulanda, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 4, 2014
Differential requirement for CCR4 and CCR7 during the development of innate and adaptive αβT cells in the adult thymusJennifer E Cowan, Nicholas I McCarthy, Sonia M Parnell, et al.
Diabetes|August 5, 2009
Common genetic variation near melatonin receptor MTNR1B contributes to raised plasma glucose and increased risk of type 2 diabetes among Indian Asians and European CaucasiansJohn C Chambers, Weihua Zhang, Delilah Zabaneh, et al.
Diabetes|April 30, 2003
Association and haplotype analysis of the insulin-degrading enzyme (IDE) gene, a strong positional and biological candidate for type 2 diabetes susceptibilityChristopher J Groves, Steven Wiltshire, Damian Smedley, et al.
Clinical Pharmacology and Therapeutics|December 24, 2010
Common nonsynonymous substitutions in SLCO1B1 predispose to statin intolerance in routinely treated individuals with type 2 diabetes: a go-DARTS studyL A Donnelly, A S F Doney, R Tavendale, et al.
BMC Medical Genomics|August 20, 2009
MicroRNA-125a is over-expressed in insulin target tissues in a spontaneous rat model of Type 2 DiabetesBlanca M Herrera, Helen E Lockstone, Jennifer M Taylor, et al.
Current Topics in Microbiology and Immunology|April 25, 2013
Mechanisms of thymus medulla development and functionGraham Anderson, Song Baik, Jennifer E Cowan, et al.
The Journal of Clinical Endocrinology and Metabolism|July 11, 2002
Delayed metabolic and thermogenic response to a mixed meal in normoglycemic European women with previous gestational diabetesEleni Kousta, Kim H Parker, Natasha J Lawrence, et al.
Plos Genetics|December 7, 2020
Common maternal and fetal genetic variants show expected polygenic effects on risk of small- or large-for-gestational-age (SGA or LGA), except in the smallest 3% of babiesRobin N Beaumont, Sarah J Kotecha, Andrew R Wood, et al.
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