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I Puscas

Showing results (11-20 of 29) with videos related to

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Experimental and Toxicologic Pathology : Official Journal of the Gesellschaft Fur Toxikologische Pathologie|November 23, 2000
Cysteamine, the most potent ulcerogenic drug known so far, powerfully activates carbonic anhydrase I, II and IV. In vitro and in vivo studiesI Puscas, M Coltau, A Maghiar, et al.
Acta Physiologica Hungarica|January 1, 1989
Effects of the synthetic PGE1 derivative misoprostol on basal, stimulated and nocturnal acid secretion in duodenal ulcer cases. Mechanism of actionG Buzás, I Puscas, F Persa, et al.
Acta Physiologica Hungarica|January 1, 1989
Prevention of non-steroidal anti-inflammatory agents induced acute gastric mucosal lesions by carbonic anhydrase inhibitors. An endoscopic studyI Puscas, A Hajdu, G Buzás, et al.
Acta Physiologica Hungarica|January 1, 1989
Carbonic anhydrase inhibitors: antisecretory and cytoprotective propertiesI Puscas, G Buzás, J C Puscas, et al.
Physiological Research|September 12, 2001
Calcium, carbonic anhydrase and gastric acid secretionI Puscas, M Coltau, M Baican, et al.
Research Communications in Molecular Pathology and Pharmacology|August 23, 2000
The inhibitory effect of diuretics on carbonic anhydrasesI Puscas, M Coltau, M Baican, et al.
Drugs Under Experimental and Clinical Research|March 14, 2000
Vasodilatory effect of diuretics is dependent on inhibition of vascular smooth muscle carbonic anhydrase by a direct mechanism of actionI Puscas, M Coltau, M Baican, et al.
Cancer Letters|August 28, 1999
Erythrocyte superoxide dismutase activity in patients with digest cancer: adjuvant diagnosis testI Puscas, M Baican, M Coltãu, et al.
Clinical and Experimental Pharmacology & Physiology|March 4, 2000
Carbonic anhydrase I inhibition by nitric oxide: implications for mediation of the hypercapnia-induced vasodilator responseI Puscas, M Coltau, G Domuta, et al.
Drugs Under Experimental and Clinical Research|June 8, 2001
Vasoconstrictive drugs increase carbonic anhydrase I in vascular smooth muscle while vasodilating drugs reduce the activity of this isozyme by a direct mechanism of actionI Puscas, M Coltau, M Baican, et al.
Pageof 3

Showing results (11-20 of 29) with videos related to

Sort By:
Pageof 3
Experimental and Toxicologic Pathology : Official Journal of the Gesellschaft Fur Toxikologische Pathologie|November 23, 2000
Cysteamine, the most potent ulcerogenic drug known so far, powerfully activates carbonic anhydrase I, II and IV. In vitro and in vivo studiesI Puscas, M Coltau, A Maghiar, et al.
Acta Physiologica Hungarica|January 1, 1989
Effects of the synthetic PGE1 derivative misoprostol on basal, stimulated and nocturnal acid secretion in duodenal ulcer cases. Mechanism of actionG Buzás, I Puscas, F Persa, et al.
Acta Physiologica Hungarica|January 1, 1989
Prevention of non-steroidal anti-inflammatory agents induced acute gastric mucosal lesions by carbonic anhydrase inhibitors. An endoscopic studyI Puscas, A Hajdu, G Buzás, et al.
Acta Physiologica Hungarica|January 1, 1989
Carbonic anhydrase inhibitors: antisecretory and cytoprotective propertiesI Puscas, G Buzás, J C Puscas, et al.
Physiological Research|September 12, 2001
Calcium, carbonic anhydrase and gastric acid secretionI Puscas, M Coltau, M Baican, et al.
Research Communications in Molecular Pathology and Pharmacology|August 23, 2000
The inhibitory effect of diuretics on carbonic anhydrasesI Puscas, M Coltau, M Baican, et al.
Drugs Under Experimental and Clinical Research|March 14, 2000
Vasodilatory effect of diuretics is dependent on inhibition of vascular smooth muscle carbonic anhydrase by a direct mechanism of actionI Puscas, M Coltau, M Baican, et al.
Cancer Letters|August 28, 1999
Erythrocyte superoxide dismutase activity in patients with digest cancer: adjuvant diagnosis testI Puscas, M Baican, M Coltãu, et al.
Clinical and Experimental Pharmacology & Physiology|March 4, 2000
Carbonic anhydrase I inhibition by nitric oxide: implications for mediation of the hypercapnia-induced vasodilator responseI Puscas, M Coltau, G Domuta, et al.
Drugs Under Experimental and Clinical Research|June 8, 2001
Vasoconstrictive drugs increase carbonic anhydrase I in vascular smooth muscle while vasodilating drugs reduce the activity of this isozyme by a direct mechanism of actionI Puscas, M Coltau, M Baican, et al.
Pageof 3