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The Journal of Experimental Medicine|November 26, 2003
Lymphotoxin is required for maintaining physiological levels of serum IgE that minimizes Th1-mediated airway inflammationHyung-Sik Kang, Sarah E Blink, Robert K Chin, et al.
The Journal of Clinical Investigation|April 16, 2013
IL-33-dependent induction of allergic lung inflammation by FcγRIII signalingMelissa Y Tjota, Jesse W Williams, Tiffany Lu, et al.
The Journal of Allergy and Clinical Immunology|December 3, 2015
Corticosteroid therapy and airflow obstruction influence the bronchial microbiome, which is distinct from that of bronchoalveolar lavage in asthmatic airwaysDarcy R Denner, Naseer Sangwan, Julia B Becker, et al.
Mucosal Immunology|June 11, 2020
Pro-lymphangiogenic VEGFR-3 signaling modulates memory T cell responses in allergic airway inflammationKatharina Maisel, Cara L Hrusch, Jorge E G Medellin, et al.
The Journal of Experimental Medicine|April 19, 2006
Fas-positive T cells regulate the resolution of airway inflammation in a murine model of asthmaJiankun Tong, Hozefa S Bandulwala, Bryan S Clay, et al.
Respiratory Research|December 10, 2022
Gut microbiota modulates bleomycin-induced acute lung injury response in miceYoung Me Yoon, Cara L Hrusch, Na Fei, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|April 16, 2013
Regulation of myofibroblast differentiation and bleomycin-induced pulmonary fibrosis by adrenomedullinJacob Kach, Nathan Sandbo, Nan Sethakorn, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|October 1, 2013
RGS3 controls T lymphocyte migration in a model of Th2-mediated airway inflammationJesse W Williams, Douglas Yau, Nan Sethakorn, et al.
Medrxiv : the Preprint Server for Health Sciences|March 4, 2025
Integration of functional genomics and statistical fine-mapping systematically characterizes adult-onset and childhood-onset asthma genetic associationsXiaoyuan Zhong, Robert Mitchell, Christine Billstrand, et al.
European Journal of Heart Failure|June 27, 2012
Elevated urinary neutrophil gelatinase-associated lipocalcin after acute heart failure treatment is associated with worsening renal function and adverse eventsSean P Collins, Kimberly W Hart, Christopher J Lindsell, et al.
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