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Ian C Forster

Showing results (41-50 of 58) with videos related to

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The Journal of General Physiology|July 5, 2024
Different fluorescent labels report distinct components of spHCN channel voltage sensor movementMagdalena N Wojciechowski, Chaseley E McKenzie, Andrew Hung, et al.
Epilepsia|August 5, 2025
Org 34167 rescues voltage dependence of mutant channels and normalizes hyperexcitability in HCN1 epilepsyLauren E Bleakley, Chaseley E McKenzie, Khaing Phyu Aung, et al.
Progress in Biophysics and Molecular Biology|May 23, 2022
Corrigendum to "Biophysical analysis of an HCN1 epilepsy variant suggests a critical role for S5 helix Met-305 in voltage sensor to pore domain coupling" [Prog. Biophys. Mol. Biol. 166 (2021) 156-172]Andrew Hung, Ian C Forster, Chaseley E Mckenzie, et al.
Progress in Biophysics and Molecular Biology|July 23, 2021
Biophysical analysis of an HCN1 epilepsy variant suggests a critical role for S5 helix Met-305 in voltage sensor to pore domain couplingAndrew Hung, Ian C Forster, Chaseley E Mckenzie, et al.
Pediatric Nephrology (Berlin, Germany)|July 6, 2016
Loss of function of NaPiIIa causes nephrocalcinosis and possibly kidney insufficiencyDganit Dinour, Miriam Davidovits, Liat Ganon, et al.
Frontiers in Pharmacology|May 26, 2023
Antidepressant-like activity of a brain penetrant HCN channel inhibitor in micePaulo Pinares-Garcia, James Spyrou, Chaseley E McKenzie, et al.
Eneuro|June 15, 2017
Shaping of Signal Transmission at the Photoreceptor Synapse by EAAT2 Glutamate TransportersStephanie Niklaus, Lucia Cadetti, Colette M Vom Berg-Maurer, et al.
The Journal of Biological Chemistry|November 10, 2018
Functional consequences of the CAPOS mutation E818K of Na<sup>+</sup>,K<sup>+</sup>-ATPaseChristian P Roenn, Melody Li, Vivien R Schack, et al.
Frontiers in Neurology|April 1, 2022
Impaired Color Recognition in <i>HCN1</i> Epilepsy: A Single Case ReportChaseley E Mckenzie, Chen-Jui Ho, Ian C Forster, et al.
Brain Communications|June 2, 2023
Cation leak: a common functional defect causing <i>HCN1</i> developmental and epileptic encephalopathyChaseley E McKenzie, Ian C Forster, Ming S Soh, et al.
Pageof 6

Showing results (41-50 of 58) with videos related to

Sort By:
Pageof 6
The Journal of General Physiology|July 5, 2024
Different fluorescent labels report distinct components of spHCN channel voltage sensor movementMagdalena N Wojciechowski, Chaseley E McKenzie, Andrew Hung, et al.
Epilepsia|August 5, 2025
Org 34167 rescues voltage dependence of mutant channels and normalizes hyperexcitability in HCN1 epilepsyLauren E Bleakley, Chaseley E McKenzie, Khaing Phyu Aung, et al.
Progress in Biophysics and Molecular Biology|May 23, 2022
Corrigendum to "Biophysical analysis of an HCN1 epilepsy variant suggests a critical role for S5 helix Met-305 in voltage sensor to pore domain coupling" [Prog. Biophys. Mol. Biol. 166 (2021) 156-172]Andrew Hung, Ian C Forster, Chaseley E Mckenzie, et al.
Progress in Biophysics and Molecular Biology|July 23, 2021
Biophysical analysis of an HCN1 epilepsy variant suggests a critical role for S5 helix Met-305 in voltage sensor to pore domain couplingAndrew Hung, Ian C Forster, Chaseley E Mckenzie, et al.
Pediatric Nephrology (Berlin, Germany)|July 6, 2016
Loss of function of NaPiIIa causes nephrocalcinosis and possibly kidney insufficiencyDganit Dinour, Miriam Davidovits, Liat Ganon, et al.
Frontiers in Pharmacology|May 26, 2023
Antidepressant-like activity of a brain penetrant HCN channel inhibitor in micePaulo Pinares-Garcia, James Spyrou, Chaseley E McKenzie, et al.
Eneuro|June 15, 2017
Shaping of Signal Transmission at the Photoreceptor Synapse by EAAT2 Glutamate TransportersStephanie Niklaus, Lucia Cadetti, Colette M Vom Berg-Maurer, et al.
The Journal of Biological Chemistry|November 10, 2018
Functional consequences of the CAPOS mutation E818K of Na<sup>+</sup>,K<sup>+</sup>-ATPaseChristian P Roenn, Melody Li, Vivien R Schack, et al.
Frontiers in Neurology|April 1, 2022
Impaired Color Recognition in <i>HCN1</i> Epilepsy: A Single Case ReportChaseley E Mckenzie, Chen-Jui Ho, Ian C Forster, et al.
Brain Communications|June 2, 2023
Cation leak: a common functional defect causing <i>HCN1</i> developmental and epileptic encephalopathyChaseley E McKenzie, Ian C Forster, Ming S Soh, et al.
Pageof 6