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Current Atherosclerosis Reports|July 7, 2023
Membrane Cholesterol Interactions with Proteins in Hypercholesterolemia-Induced Endothelial DysfunctionIbra S Fancher, Irena Levitan
Journal of Visualized Experiments : Jove|August 20, 2019
Electrophysiological Recordings of Single-cell Ion Currents Under Well-defined Shear StressIbra S Fancher, Irena Levitan
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|May 31, 2019
Shear-Stress Sensitive Inwardly-Rectifying K<sup>+</sup> Channels Regulate Developmental Retinal Angiogenesis by Vessel RegressionEvgenii Boriushkin, Ibra S Fancher, Irena Levitan
Journal of Biomechanics|June 30, 2018
Comparative analysis of endothelial cell and sub-endothelial cell elastic moduli in young and aged mice: Role of CD36Elizabeth Le Master, Ibra S Fancher, James Lee, et al.
American Journal of Physiology. Heart and Circulatory Physiology|December 10, 2021
Differential effects of obesity on visceral versus subcutaneous adipose arteries: role of shear-activated Kir2.1 and alterations to the glycocalyxSang Joon Ahn, Elizabeth Le Master, James C Lee, et al.
Hypertension (Dallas, Tex. : 1979)|November 17, 2021
Cholesterol-Induced Suppression of Endothelial Kir Channels Is a Driver of Impairment of Arteriolar Flow-Induced Vasodilation in HumansSang Joon Ahn, Ibra S Fancher, Sara T Granados, et al.
The Journal of Physiology|November 19, 2016
Inwardly rectifying K<sup>+</sup> channels are major contributors to flow-induced vasodilatation in resistance arteriesSang Joon Ahn, Ibra S Fancher, Jing-Tan Bian, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
Protection against diet-induced obesity by a single-point mutation in Kir2.1 channelsKatie M Beverley, Marcos D Munoz, Sang Joon Ahn, et al.
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