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Ida G Lunde

Showing results (41-50 of 60) with videos related to

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Journal of Molecular Medicine (Berlin, Germany)|March 31, 2017
Wnt5a is elevated in heart failure and affects cardiac fibroblast functionAurelija Abraityte, Leif E Vinge, Erik T Askevold, et al.
The FEBS Journal|February 5, 2013
Innate immune signaling induces expression and shedding of the heparan sulfate proteoglycan syndecan-4 in cardiac fibroblasts and myocytes, affecting inflammation in the pressure-overloaded heartMari E Strand, Kate M Herum, Zaheer A Rana, et al.
Journal of the American Heart Association|February 1, 2020
Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved OsteopontinKate M Herum, Andreas Romaine, Ariel Wang, et al.
American Journal of Physiology. Cell Physiology|March 14, 2026
Effects of proANP<sub>31-67</sub> in a preclinical model of uninephrectomy and cardiac ischemia/reperfusion injury: cardiac remodeling and tissue biochemical profilingGustavo Jose Justo Silva, Sara Stefani, Reza Parvan, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 8, 2019
Increased Complement Factor B and Bb Levels Are Associated with Mortality in Patients with Severe Aortic StenosisNegar Shahini, Thor Ueland, Andreas Auensen, et al.
Plos One|December 14, 2011
Syndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathwayAlexandra V Finsen, Ida G Lunde, Ivar Sjaastad, et al.
Science Translational Medicine|January 25, 2019
Hypertrophic cardiomyopathy mutations in <i>MYBPC3</i> dysregulate myosinChristopher N Toepfer, Hiroko Wakimoto, Amanda C Garfinkel, et al.
Circulation Research|July 4, 2023
Stretch Harmonizes Sarcomere Strain Across the CardiomyocyteJia Li, Joakim Sundnes, Yufeng Hou, et al.
Communications Biology|December 20, 2022
ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overloadKaroline B Rypdal, A Olav Melleby, Emma L Robinson, et al.
European Journal of Heart Failure|June 5, 2024
Mechanisms of myocardial reverse remodelling and its clinical significance: A scientific statement of the ESC Working Group on Myocardial FunctionInês Falcão-Pires, Ana Filipa Ferreira, Fábio Trindade, et al.
Pageof 6

Showing results (41-50 of 60) with videos related to

Sort By:
Pageof 6
Journal of Molecular Medicine (Berlin, Germany)|March 31, 2017
Wnt5a is elevated in heart failure and affects cardiac fibroblast functionAurelija Abraityte, Leif E Vinge, Erik T Askevold, et al.
The FEBS Journal|February 5, 2013
Innate immune signaling induces expression and shedding of the heparan sulfate proteoglycan syndecan-4 in cardiac fibroblasts and myocytes, affecting inflammation in the pressure-overloaded heartMari E Strand, Kate M Herum, Zaheer A Rana, et al.
Journal of the American Heart Association|February 1, 2020
Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved OsteopontinKate M Herum, Andreas Romaine, Ariel Wang, et al.
American Journal of Physiology. Cell Physiology|March 14, 2026
Effects of proANP<sub>31-67</sub> in a preclinical model of uninephrectomy and cardiac ischemia/reperfusion injury: cardiac remodeling and tissue biochemical profilingGustavo Jose Justo Silva, Sara Stefani, Reza Parvan, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 8, 2019
Increased Complement Factor B and Bb Levels Are Associated with Mortality in Patients with Severe Aortic StenosisNegar Shahini, Thor Ueland, Andreas Auensen, et al.
Plos One|December 14, 2011
Syndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathwayAlexandra V Finsen, Ida G Lunde, Ivar Sjaastad, et al.
Science Translational Medicine|January 25, 2019
Hypertrophic cardiomyopathy mutations in <i>MYBPC3</i> dysregulate myosinChristopher N Toepfer, Hiroko Wakimoto, Amanda C Garfinkel, et al.
Circulation Research|July 4, 2023
Stretch Harmonizes Sarcomere Strain Across the CardiomyocyteJia Li, Joakim Sundnes, Yufeng Hou, et al.
Communications Biology|December 20, 2022
ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overloadKaroline B Rypdal, A Olav Melleby, Emma L Robinson, et al.
European Journal of Heart Failure|June 5, 2024
Mechanisms of myocardial reverse remodelling and its clinical significance: A scientific statement of the ESC Working Group on Myocardial FunctionInês Falcão-Pires, Ana Filipa Ferreira, Fábio Trindade, et al.
Pageof 6