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Frontiers in Pharmacology|January 9, 2019
The Autophagy Inhibitor Spautin-1 Antagonizes Rescue of Mutant CFTR Through an Autophagy-Independent and USP13-Mediated MechanismEmanuela Pesce, Elvira Sondo, Loretta Ferrera, et al.
Neurobiology of Disease|December 1, 2025
Modeling Mowat-Wilson syndrome with patient iPSCs reveals transcriptional and phenotypic defects in neural progenitorsIlaria Musante, Giulia Gorrieri, Serena Tamburro, et al.
Journal of Cystic Fibrosis : Official Journal of the European Cystic Fibrosis Society|June 16, 2018
Combination potentiator ('co-potentiator') therapy for CF caused by CFTR mutants, including N1303K, that are poorly responsive to single potentiatorsPuay-Wah Phuan, Jung-Ho Son, Joseph-Anthony Tan, et al.
Human Mutation|March 10, 2019
Two CFTR mutations within codon 970 differently impact on the chloride channel functionalityFelice Amato, Paolo Scudieri, Ilaria Musante, et al.
International Journal of Molecular Sciences|June 2, 2021
Partial Rescue of F508del-CFTR Stability and Trafficking Defects by Double Corrector TreatmentValeria Capurro, Valeria Tomati, Elvira Sondo, et al.
Scientific Reports|May 10, 2023
Novel tricyclic pyrrolo-quinolines as pharmacological correctors of the mutant CFTR chloride channelMario Renda, Marilia Barreca, Anna Borrelli, et al.
Scientific Reports|October 28, 2016
Goblet Cell Hyperplasia Requires High Bicarbonate Transport To Support Mucin ReleaseGiulia Gorrieri, Paolo Scudieri, Emanuela Caci, et al.
BMC Medical Genetics|November 26, 2016
Assessment of copy number variations in 120 patients with Poland syndromeCarlotta Maria Vaccari, Elisa Tassano, Michele Torre, et al.
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