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Ildem Akerman

Showing results (11-20 of 28) with videos related to

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The Journal of Biological Chemistry|July 26, 2011
Hepatic nuclear factor 1alpha (HNF1alpha) dysfunction down-regulates X-box-binding protein 1 (XBP1) and sensitizes beta-cells to endoplasmic reticulum stressClare L Kirkpatrick, Andreas Wiederkehr, Mathurin Baquié, et al.
Nature Genetics|December 14, 2011
GATA6 haploinsufficiency causes pancreatic agenesis in humansHana Lango Allen, Sarah E Flanagan, Charles Shaw-Smith, et al.
Cell Reports|April 14, 2021
Neonatal diabetes mutations disrupt a chromatin pioneering function that activates the human insulin geneIldem Akerman, Miguel Angel Maestro, Elisa De Franco, et al.
Nature Communications|September 22, 2020
A predictable conserved DNA base composition signature defines human core DNA replication originsIldem Akerman, Bahar Kasaai, Alina Bazarova, et al.
Biorxiv : the Preprint Server for Biology|June 5, 2026
High-purity stem cell-derived β-cells recapitulate key transcriptional and functional features of human isletsRémi B Fiancette, Junyue Huang, Christine Stephens, et al.
JCI Insight|July 15, 2021
Prolyl-4-hydroxylase 3 maintains β cell glucose metabolism during fatty acid excess in miceDaniela Nasteska, Federica Cuozzo, Katrina Viloria, et al.
JCI Insight|April 13, 2021
Systemic and adipocyte transcriptional and metabolic dysregulation in idiopathic intracranial hypertensionConnar Sj Westgate, Hannah F Botfield, Zerin Alimajstorovic, et al.
Nature Communications|January 30, 2021
PDX1<sup>LOW</sup> MAFA<sup>LOW</sup> β-cells contribute to islet function and insulin releaseDaniela Nasteska, Nicholas H F Fine, Fiona B Ashford, et al.
Nature Communications|July 21, 2021
Author Correction: PDX1<sup>LOW</sup> MAFA<sup>LOW</sup> β-cells contribute to islet function and insulin releaseDaniela Nasteska, Nicholas H F Fine, Fiona B Ashford, et al.
Nature Communications|July 2, 2025
A heterozygous CEBPA mutation disrupting the bZIP domain in a RUNX1 and SRSF2 mutational background causes MDS disease progressionRuba Almaghrabi, Yara Alyahyawi, Peter Keane, et al.
Pageof 3

Showing results (11-20 of 28) with videos related to

Sort By:
Pageof 3
The Journal of Biological Chemistry|July 26, 2011
Hepatic nuclear factor 1alpha (HNF1alpha) dysfunction down-regulates X-box-binding protein 1 (XBP1) and sensitizes beta-cells to endoplasmic reticulum stressClare L Kirkpatrick, Andreas Wiederkehr, Mathurin Baquié, et al.
Nature Genetics|December 14, 2011
GATA6 haploinsufficiency causes pancreatic agenesis in humansHana Lango Allen, Sarah E Flanagan, Charles Shaw-Smith, et al.
Cell Reports|April 14, 2021
Neonatal diabetes mutations disrupt a chromatin pioneering function that activates the human insulin geneIldem Akerman, Miguel Angel Maestro, Elisa De Franco, et al.
Nature Communications|September 22, 2020
A predictable conserved DNA base composition signature defines human core DNA replication originsIldem Akerman, Bahar Kasaai, Alina Bazarova, et al.
Biorxiv : the Preprint Server for Biology|June 5, 2026
High-purity stem cell-derived β-cells recapitulate key transcriptional and functional features of human isletsRémi B Fiancette, Junyue Huang, Christine Stephens, et al.
JCI Insight|July 15, 2021
Prolyl-4-hydroxylase 3 maintains β cell glucose metabolism during fatty acid excess in miceDaniela Nasteska, Federica Cuozzo, Katrina Viloria, et al.
JCI Insight|April 13, 2021
Systemic and adipocyte transcriptional and metabolic dysregulation in idiopathic intracranial hypertensionConnar Sj Westgate, Hannah F Botfield, Zerin Alimajstorovic, et al.
Nature Communications|January 30, 2021
PDX1<sup>LOW</sup> MAFA<sup>LOW</sup> β-cells contribute to islet function and insulin releaseDaniela Nasteska, Nicholas H F Fine, Fiona B Ashford, et al.
Nature Communications|July 21, 2021
Author Correction: PDX1<sup>LOW</sup> MAFA<sup>LOW</sup> β-cells contribute to islet function and insulin releaseDaniela Nasteska, Nicholas H F Fine, Fiona B Ashford, et al.
Nature Communications|July 2, 2025
A heterozygous CEBPA mutation disrupting the bZIP domain in a RUNX1 and SRSF2 mutational background causes MDS disease progressionRuba Almaghrabi, Yara Alyahyawi, Peter Keane, et al.
Pageof 3