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Journal of Cellular and Molecular Medicine|February 22, 2012
Inflammation in atherosclerosis: a cause or a result of vascular disorders?Ileana Manduteanu, Maya SimionescuCurrent Medicinal Chemistry|November 25, 2016
Emerging Nanocarriers-based Approaches to Diagnose and Red uce Vascular Inflammation in AtherosclerosisManuela Calin, Ileana ManduteanuJournal of Cellular and Molecular Medicine|September 4, 2025
Recent Advances in Deciphering Normal and Diseased Aortic Valve Biology Using Transcriptomic TechnologiesMonica Madalina Tucureanu, Ileana ManduteanuCritical Reviews in Eukaryotic Gene Expression|November 19, 2014
Molecular and functional interactions among monocytes/macrophages and smooth muscle cells and their relevance for atherosclerosisElena Butoi, Ana Maria Gan, Ileana ManduteanuJournal of Cellular and Molecular Medicine|September 25, 2021
Aortic valve disease in diabetes: Molecular mechanisms and novel therapiesIleana Manduteanu, Dan Simionescu, Agneta Simionescu, et al.Vascular Pharmacology|March 6, 2003
Binding and uptake of transferrin-bound liposomes targeted to transferrin receptors of endothelial cellsManuela Voinea, Elena Dragomir, Ileana Manduteanu, et al.Vascular Pharmacology|April 8, 2006
Aspirin and PPAR-alpha activators inhibit monocyte chemoattractant protein-1 expression induced by high glucose concentration in human endothelial cellsElena Dragomir, Magdalena Tircol, Ileana Manduteanu, et al.Pharmaceutical Research|August 10, 2005
Immunoliposomes directed toward VCAM-1 interact specifically with activated endothelial cells--a potential tool for specific drug deliveryManuela Voinea, Ileana Manduteanu, Elena Dragomir, et al.Pharmacology|January 30, 2007
Enoxaparin reduces H2O2-induced activation of human endothelial cells by a mechanism involving cell adhesion molecules and nuclear transcription factorsIleana Manduteanu, Elena Dragomir, Manuela Voinea, et al.Journal of Diabetes and Its Complications|September 1, 2004
Aspirin rectifies calcium homeostasis, decreases reactive oxygen species, and increases NO production in high glucose-exposed human endothelial cellsElena Dragomir, Ileana Manduteanu, Manuela Voinea, et al.Pageof 5