Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Inger Lauritzen

Showing results (11-20 of 29) with videos related to

Pageof 3
Sort By:
Neurobiology of Aging|February 6, 2014
Study on Aβ34 biology and detection in transgenic mice brainsCéline Caillava, Sébastien Ranaldi, Inger Lauritzen, et al.
The Journal of Biological Chemistry|June 5, 2003
K+-dependent cerebellar granule neuron apoptosis. Role of task leak K+ channelsInger Lauritzen, Marc Zanzouri, Eric Honoré, et al.
Translational Neurodegeneration|December 13, 2019
Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse modelsInger Lauritzen, Anaïs Bécot, Alexandre Bourgeois, et al.
Journal of Extracellular Vesicles|January 16, 2025
Presenilins as hub proteins controlling the endocytic and autophagic pathways and small extracellular vesicle secretionInger Lauritzen, Anaïs Bini, Anaïs Bécot, et al.
Journal of Alzheimer'S Disease : JAD|March 13, 2012
Evidence that the amyloid-β protein precursor intracellular domain, AICD, derives from β-secretase-generated C-terminal fragmentBrice Flammang, Raphaëlle Pardossi-Piquard, Jean Sevalle, et al.
Cells|May 16, 2020
The Transcription Factor EB Reduces the Intraneuronal Accumulation of the Beta-Secretase-Derived APP Fragment C99 in Cellular and Mouse Alzheimer’s Disease ModelsAnaïs Bécot, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, et al.
Cell Death and Differentiation|January 21, 2018
Nuclear p53-mediated repression of autophagy involves PINK1 transcriptional down-regulationThomas Goiran, Eric Duplan, Lila Rouland, et al.
Acta Neuropathologica|May 4, 2016
Intraneuronal aggregation of the β-CTF fragment of APP (C99) induces Aβ-independent lysosomal-autophagic pathologyInger Lauritzen, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, et al.
Acta Neuropathologica|August 4, 2023
Correction to: Intraneuronal aggregation of the β‑CTF fragment of APP (C99) induces Aβ‑independent lysosomal-autophagic pathologyInger Lauritzen, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, et al.
Cellular and Molecular Life Sciences : CMLS|March 17, 2023
The η-secretase-derived APP fragment ηCTF is localized in Golgi, endosomes and extracellular vesicles and contributes to Aβ productionElissa Afram, Inger Lauritzen, Alexandre Bourgeois, et al.
Pageof 3

Showing results (11-20 of 29) with videos related to

Sort By:
Pageof 3
Neurobiology of Aging|February 6, 2014
Study on Aβ34 biology and detection in transgenic mice brainsCéline Caillava, Sébastien Ranaldi, Inger Lauritzen, et al.
The Journal of Biological Chemistry|June 5, 2003
K+-dependent cerebellar granule neuron apoptosis. Role of task leak K+ channelsInger Lauritzen, Marc Zanzouri, Eric Honoré, et al.
Translational Neurodegeneration|December 13, 2019
Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse modelsInger Lauritzen, Anaïs Bécot, Alexandre Bourgeois, et al.
Journal of Extracellular Vesicles|January 16, 2025
Presenilins as hub proteins controlling the endocytic and autophagic pathways and small extracellular vesicle secretionInger Lauritzen, Anaïs Bini, Anaïs Bécot, et al.
Journal of Alzheimer'S Disease : JAD|March 13, 2012
Evidence that the amyloid-β protein precursor intracellular domain, AICD, derives from β-secretase-generated C-terminal fragmentBrice Flammang, Raphaëlle Pardossi-Piquard, Jean Sevalle, et al.
Cells|May 16, 2020
The Transcription Factor EB Reduces the Intraneuronal Accumulation of the Beta-Secretase-Derived APP Fragment C99 in Cellular and Mouse Alzheimer’s Disease ModelsAnaïs Bécot, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, et al.
Cell Death and Differentiation|January 21, 2018
Nuclear p53-mediated repression of autophagy involves PINK1 transcriptional down-regulationThomas Goiran, Eric Duplan, Lila Rouland, et al.
Acta Neuropathologica|May 4, 2016
Intraneuronal aggregation of the β-CTF fragment of APP (C99) induces Aβ-independent lysosomal-autophagic pathologyInger Lauritzen, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, et al.
Acta Neuropathologica|August 4, 2023
Correction to: Intraneuronal aggregation of the β‑CTF fragment of APP (C99) induces Aβ‑independent lysosomal-autophagic pathologyInger Lauritzen, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, et al.
Cellular and Molecular Life Sciences : CMLS|March 17, 2023
The η-secretase-derived APP fragment ηCTF is localized in Golgi, endosomes and extracellular vesicles and contributes to Aβ productionElissa Afram, Inger Lauritzen, Alexandre Bourgeois, et al.
Pageof 3