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Ion C Cirstea

Showing results (1-10 of 15) with videos related to

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Trends in Cell Biology|November 25, 2022
Glucocorticoid receptor and RAS: an unexpected couple in cancerIon C Cirstea, Herwig P Moll, Jan Tuckermann
Pharmaceuticals (Basel, Switzerland)|May 10, 2019
Deregulation of Hepatic Mek1/2⁻Erk1/2 Signaling Module in Iron Overload ConditionsNaveen Kumar Tangudu, Nils Buth, Pavel Strnad, et al.
The Journal of Biological Chemistry|April 2, 2011
Mechanistic insights into specificity, activity, and regulatory elements of the regulator of G-protein signaling (RGS)-containing Rho-specific guanine nucleotide exchange factors (GEFs) p115, PDZ-RhoGEF (PRG), and leukemia-associated RhoGEF (LARG)Mamta Jaiswal, Lothar Gremer, Radovan Dvorsky, et al.
Human Molecular Genetics|October 13, 2012
Diverging gain-of-function mechanisms of two novel KRAS mutations associated with Noonan and cardio-facio-cutaneous syndromesIon C Cirstea, Lothar Gremer, Radovan Dvorsky, et al.
Human Mutation|October 16, 2010
Germline KRAS mutations cause aberrant biochemical and physical properties leading to developmental disordersLothar Gremer, Torsten Merbitz-Zahradnik, Radovan Dvorsky, et al.
Endocrine|September 16, 2021
A novel in vitro assay to study chondrocyte-to-osteoblast transdifferentiationMiriam E A Tschaffon, Stefan O Reber, Astrid Schoppa, et al.
Plos One|July 12, 2014
Liposome reconstitution and modulation of recombinant prenylated human Rac1 by GEFs, GDI1 and Pak1Si-Cai Zhang, Lothar Gremer, Henrike Heise, et al.
The Journal of Biological Chemistry|May 6, 2015
The Function of Embryonic Stem Cell-expressed RAS (E-RAS), a Unique RAS Family Member, Correlates with Its Additional Motifs and Its Structural PropertiesSaeideh Nakhaei-Rad, Hossein Nakhaeizadeh, Claus Kordes, et al.
Human Molecular Genetics|November 28, 2018
Dominant Noonan syndrome-causing LZTR1 mutations specifically affect the Kelch domain substrate-recognition surface and enhance RAS-MAPK signalingMarialetizia Motta, Miray Fidan, Emanuele Bellacchio, et al.
Human Mutation|April 9, 2017
Aberrant HRAS transcript processing underlies a distinctive phenotype within the RASopathy clinical spectrumFrancesca Pantaleoni, Dorit Lev, Ion C Cirstea, et al.
Pageof 2

Showing results (1-10 of 15) with videos related to

Sort By:
Pageof 2
Trends in Cell Biology|November 25, 2022
Glucocorticoid receptor and RAS: an unexpected couple in cancerIon C Cirstea, Herwig P Moll, Jan Tuckermann
Pharmaceuticals (Basel, Switzerland)|May 10, 2019
Deregulation of Hepatic Mek1/2⁻Erk1/2 Signaling Module in Iron Overload ConditionsNaveen Kumar Tangudu, Nils Buth, Pavel Strnad, et al.
The Journal of Biological Chemistry|April 2, 2011
Mechanistic insights into specificity, activity, and regulatory elements of the regulator of G-protein signaling (RGS)-containing Rho-specific guanine nucleotide exchange factors (GEFs) p115, PDZ-RhoGEF (PRG), and leukemia-associated RhoGEF (LARG)Mamta Jaiswal, Lothar Gremer, Radovan Dvorsky, et al.
Human Molecular Genetics|October 13, 2012
Diverging gain-of-function mechanisms of two novel KRAS mutations associated with Noonan and cardio-facio-cutaneous syndromesIon C Cirstea, Lothar Gremer, Radovan Dvorsky, et al.
Human Mutation|October 16, 2010
Germline KRAS mutations cause aberrant biochemical and physical properties leading to developmental disordersLothar Gremer, Torsten Merbitz-Zahradnik, Radovan Dvorsky, et al.
Endocrine|September 16, 2021
A novel in vitro assay to study chondrocyte-to-osteoblast transdifferentiationMiriam E A Tschaffon, Stefan O Reber, Astrid Schoppa, et al.
Plos One|July 12, 2014
Liposome reconstitution and modulation of recombinant prenylated human Rac1 by GEFs, GDI1 and Pak1Si-Cai Zhang, Lothar Gremer, Henrike Heise, et al.
The Journal of Biological Chemistry|May 6, 2015
The Function of Embryonic Stem Cell-expressed RAS (E-RAS), a Unique RAS Family Member, Correlates with Its Additional Motifs and Its Structural PropertiesSaeideh Nakhaei-Rad, Hossein Nakhaeizadeh, Claus Kordes, et al.
Human Molecular Genetics|November 28, 2018
Dominant Noonan syndrome-causing LZTR1 mutations specifically affect the Kelch domain substrate-recognition surface and enhance RAS-MAPK signalingMarialetizia Motta, Miray Fidan, Emanuele Bellacchio, et al.
Human Mutation|April 9, 2017
Aberrant HRAS transcript processing underlies a distinctive phenotype within the RASopathy clinical spectrumFrancesca Pantaleoni, Dorit Lev, Ion C Cirstea, et al.
Pageof 2