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The Journal of Biological Chemistry|June 6, 2007
Accelerating amyloid-beta fibrillization reduces oligomer levels and functional deficits in Alzheimer disease mouse modelsIrene H Cheng, Kimberly Scearce-Levie, Justin Legleiter, et al.Nature Neuroscience|October 22, 2008
Phospholipase A2 reduction ameliorates cognitive deficits in a mouse model of Alzheimer's diseaseRene O Sanchez-Mejia, John W Newman, Sandy Toh, et al.Plos One|May 5, 2012
Amyloid-beta (Aβ) D7H mutation increases oligomeric Aβ42 and alters properties of Aβ-zinc/copper assembliesWei-Ting Chen, Chen-Jee Hong, Ya-Tzu Lin, et al.Plos One|December 15, 2010
ENU mutagenesis identifies mice with morbid obesity and severe hyperinsulinemia caused by a novel mutation in leptinChen-Jee Hong, Pei-Jane Tsai, Chih-Ya Cheng, et al.Nature Communications|September 13, 2014
Full-length TDP-43 forms toxic amyloid oligomers that are present in frontotemporal lobar dementia-TDP patientsYu-Sheng Fang, Kuen-Jer Tsai, Yu-Jen Chang, et al.Pageof 3